Immunology Lecture 11 Part 8 Genetics of Immunoglobulin Diversity
This video and its companions are uploaded on http://usmlestrategy99.com/usmle-step-1/immunology/itemlist/category/2-immunology-videos) Immunoglobulins are ...

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Immunology Lecture 11 Part 8 Genetics of Immunoglobulin Diversity - Video

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Philosopher Dissects White Guilt - #39;Everything Hinges On Whether It Genetics or Environmental #39;
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Philosopher Dissects White Guilt - 'Everything Hinges On Whether It Genetics or Environmental' - Video

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Myriad Genetics is up sharply since reporting a strong quarter last week, and traders are betting on more gains ahead.

Nearly 4,000 August 33 calls traded in a strong buying pattern this morning, led by a print of 3,001 contracts that went for $3.75, according to optionMONSTER's Heat Seeker system. These are clearly fresh purchases, as the volume was well above the strike's open interest of 1,656 contracts before the session began.

MYGN is up fractionally at $33.19 in midday trading. The shares gapped up from the $28 level after the medical-diagnostics company posted earnings and revenues estimates while raising guidance last Tuesday.

Today's long calls , which lock in the price where the stock can be bought no matter how far it might rise, are looking for MYGN to rally roughly 11 percent or more by expiration in mid-August. But these calls, which will track the share price closely because they are already in the money , will rapidly lose value if the stock doesn't rise. (See our Education section)

Overall option volume in the name today is already almost 6 times its full-session average. Calls outnumber puts by 16 go 1, a reflection of the bullish sentiment.

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Bulls show faith in Myriad Genetics

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MENAFN - AFP - 12/05/2013

(MENAFN - AFP) Separate studies of the human genome have found tantalising clues to the inherited causes of testicular cancer and non-inherited causes of congenital heart disease, journals reported on Sunday.University of Pennsylvania researchers looked at the DNA of more than 13,000 men, comparing the DNA code of those with testicular cancer -- the commonest form of cancer diagnosed among young men today -- against men who were otherwise healthy.They found four new variants that increase the risk of this disease, bringing the tally of known mutations to 17, according to research reported in Nature Genetics.Meanwhile, investigators at the Yale School of Medicine found a clutch of gene mutations, absent in parents but found in their offspring, which account for at least 10 percent of cases of severe congenital heart disease, a birth defect that afflicts nearly one percent of babies."Most interestingly, the set of genes mutated in congenital heart disease unexpectedly overlapped with genes and pathways mutated in autism," said Richard Lifton, a professor of genetics."These findings suggest there may be common pathways that underlie a wide range of common congenital diseases."The study appears in the journal Nature.Genomics is one of the fastest-moving areas of medical research.Identifying genetic signatures associated with disease opens up the prospect of DNA tests to identify people most at risk. They also throw open avenues of research to block or reverse the disease.

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Gene clues for testicular cancer, heart defect

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Newswise PHILADELPHIAA new study looking at the genomes of more than 13,000 men identified four new genetic variants associated with an increased risk of testicular cancer, the most commonly diagnosed type in young men today. The findings from this first-of-its-kind meta-analysis were reported online May 12 in Nature Genetics by researchers at the Perelman School of Medicine at the University of Pennsylvania.

The discovery of these genetic variationschromosomal typos, so to speakcould ultimately help researchers better understand which men are at high risk and allow for early detection or prevention of the disease.

As we continue to cast a wider net, we identify additional genetic risk factors, which point to new mechanisms for disease, said Katherine L. Nathanson, MD, associate professor in the division of Translational Medicine and Human Genetics within the department of Medicine. Certain chromosomal regions, what we call loci, are tied into testicular cancer susceptibility, and represent a promising path to stratifying patients into risk groupsfor a disease we know is highly heritable.

Tapping into three genome-wide association studies (GWAS), the researchers, including Peter A. Kanetsky, PhD, MPH, an associate professor in the department of Biostatistics and Epidemiology, analyzed 931 affected individuals and 1,975 controls and confirmed the results in an additional 3,211 men with cancer and 7,591 controls. The meta-analysis revealed that testicular germ cell tumor (TGCT) risk was significantly associated with markers at four loci4q22, 7q22, 16q22.3, and 17q22, none of which have been identified in other cancers. Additionally, these loci pose a higher risk than the vast majority of other loci identified for some common cancers, such as breast and prostate.

This brings the number of genomic regions associated with testicular cancer up to 17including eight new ones reported in another study in this issue of Nature Genetics.

Testicular cancer is relatively rare; however, incidence rates have doubled in the past 40 years. It is also highly heritable. If a man has a father or son with testicular cancer, he has a four-to six-fold higher risk of developing it compared to a man with no family history. That increases to an eight-to 10-fold higher risk if the man has a brother with testicular cancer.

Given this, researchers continue to investigate genetic variants and their association with cancer.

In 2009, Dr. Nathanson and colleagues uncovered variation around two genesKITLG and SPRY4found to be associated with an increased risk of testicular cancer. The two variants were the first striking genetic risk factors found for this disease at the time. Since then, several more variants have been discovered, but only through single GWAS studies.

This analysis is the first to bring several groups of data together to identify loci associated with disease, said Dr. Nathanson, and represent the power of combining multiple GWAS to better identify genetic risk factors that failed to reach genome-wide significance in single studies.

The team also explains how the variants associated with increased cancer risk are the same genes associated with chromosomal segregation. The variants are also found near genes important for germ cell development. These data strongly supports the notion that testicular cancer is a disorder of germ cell development and maturation.

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Researchers Identify Four New Genetic Risk Factors for Testicular Cancer

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May 12, 2013 A new study looking at the genomes of more than 13,000 men identified four new genetic variants associated with an increased risk of testicular cancer, the most commonly diagnosed type in young men today. The findings from this first-of-its-kind meta-analysis were reported online May 12 in Nature Genetics by researchers at the Perelman School of Medicine at the University of Pennsylvania.

The discovery of these genetic variations -- chromosomal "typos," so to speak -- could ultimately help researchers better understand which men are at high risk and allow for early detection or prevention of the disease.

"As we continue to cast a wider net, we identify additional genetic risk factors, which point to new mechanisms for disease," said Katherine L. Nathanson, MD, associate professor in the division of Translational Medicine and Human Genetics within the department of Medicine. "Certain chromosomal regions, what we call loci, are tied into testicular cancer susceptibility, and represent a promising path to stratifying patients into risk groups -- for a disease we know is highly heritable."

Tapping into three genome-wide association studies (GWAS), the researchers, including Peter A. Kanetsky, PhD, MPH, an associate professor in the department of Biostatistics and Epidemiology, analyzed 931 affected individuals and 1,975 controls and confirmed the results in an additional 3,211 men with cancer and 7,591 controls. The meta-analysis revealed that testicular germ cell tumor (TGCT) risk was significantly associated with markers at four loci -- 4q22, 7q22, 16q22.3, and 17q22, none of which have been identified in other cancers. Additionally, these loci pose a higher risk than the vast majority of other loci identified for some common cancers, such as breast and prostate.

This brings the number of genomic regions associated with testicular cancer up to 17 -- including eight new ones reported in another study in this issue of Nature Genetics.

Testicular cancer is relatively rare; however, incidence rates have doubled in the past 40 years. It is also highly heritable. If a man has a father or son with testicular cancer, he has a four-to six-fold higher risk of developing it compared to a man with no family history. That increases to an eight-to 10-fold higher risk if the man has a brother with testicular cancer.

Given this, researchers continue to investigate genetic variants and their association with cancer.

In 2009, Dr. Nathanson and colleagues uncovered variation around two genes -- KITLG and SPRY4 -- found to be associated with an increased risk of testicular cancer. The two variants were the first striking genetic risk factors found for this disease at the time. Since then, several more variants have been discovered, but only through single GWAS studies.

"This analysis is the first to bring several groups of data together to identify loci associated with disease," said Dr. Nathanson, "and represent the power of combining multiple GWAS to better identify genetic risk factors that failed to reach genome-wide significance in single studies."

The team also explains how the variants associated with increased cancer risk are the same genes associated with chromosomal segregation. The variants are also found near genes important for germ cell development. These data strongly supports the notion that testicular cancer is a disorder of germ cell development and maturation.

Continue reading here:
Four new genetic risk factors for testicular cancer identified

Recommendation and review posted by Bethany Smith

Public release date: 12-May-2013 [ | E-mail | Share ]

Contact: Steve Graff stephen.graff@uphs.upenn.edu 215-349-5653 University of Pennsylvania School of Medicine

PHILADELPHIAA new study looking at the genomes of more than 13,000 men identified four new genetic variants associated with an increased risk of testicular cancer, the most commonly diagnosed type in young men today. The findings from this first-of-its-kind meta-analysis were reported online May 12 in Nature Genetics by researchers at the Perelman School of Medicine at the University of Pennsylvania.

The discovery of these genetic variationschromosomal "typos," so to speakcould ultimately help researchers better understand which men are at high risk and allow for early detection or prevention of the disease.

"As we continue to cast a wider net, we identify additional genetic risk factors, which point to new mechanisms for disease," said Katherine L. Nathanson, MD, associate professor in the division of Translational Medicine and Human Genetics within the department of Medicine. "Certain chromosomal regions, what we call loci, are tied into testicular cancer susceptibility, and represent a promising path to stratifying patients into risk groupsfor a disease we know is highly heritable."

Tapping into three genome-wide association studies (GWAS), the researchers, including Peter A. Kanetsky, PhD, MPH, an associate professor in the department of Biostatistics and Epidemiology, analyzed 931 affected individuals and 1,975 controls and confirmed the results in an additional 3,211 men with cancer and 7,591 controls. The meta-analysis revealed that testicular germ cell tumor (TGCT) risk was significantly associated with markers at four loci4q22, 7q22, 16q22.3, and 17q22, none of which have been identified in other cancers. Additionally, these loci pose a higher risk than the vast majority of other loci identified for some common cancers, such as breast and prostate.

This brings the number of genomic regions associated with testicular cancer up to 17including eight new ones reported in another study in this issue of Nature Genetics.

Testicular cancer is relatively rare; however, incidence rates have doubled in the past 40 years. It is also highly heritable. If a man has a father or son with testicular cancer, he has a four-to six-fold higher risk of developing it compared to a man with no family history. That increases to an eight-to 10-fold higher risk if the man has a brother with testicular cancer.

Given this, researchers continue to investigate genetic variants and their association with cancer.

In 2009, Dr. Nathanson and colleagues uncovered variation around two genesKITLG and SPRY4found to be associated with an increased risk of testicular cancer. The two variants were the first striking genetic risk factors found for this disease at the time. Since then, several more variants have been discovered, but only through single GWAS studies.

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Penn Medicine researchers identify 4 new genetic risk factors for testicular cancer

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2011 Champions of Genetics
In May of 2011, the Canadian Gene Cure Foundation celebrated it #39;s first Champions of Genetics and announced it #39;s first three award recipients. This exciting ...

By: GeneCure

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2011 Champions of Genetics - Video

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Day 1 Ocean Grown Genetics Pheno hunt..
All 6 merlot og beans popd..5 herijuana og beans popd..5 alien abduction beans popd.. Their all n happy frogs soil under 1 t5 ..stay tuned for hopefully week...

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Day 1 Ocean Grown Genetics Pheno hunt.. - Video

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Genetics Study Shows Europe is One Big Family
A mitochondrial DNA study found if lineages are traced back just 1000 years, nearly all Europeans turn out to be related.

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Genetics Study Shows Europe is One Big Family - Video

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alta genetics youtube)

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alta genetics youtube) - Video

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Let #39;s Play The Sims 3 - Perfect Genetics - Episode 5 - Rummaging
With the arrival of Addison, Heather takes some time off and attends to the needs of her daughter. Not convinced that she is going to have the proper genetic...

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Immunology Lecture 11 Part 7 Genetics of Immunoglobulin Diversity
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By: Mobeen Syed

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Immunology Lecture 11 Part 7 Genetics of Immunoglobulin Diversity - Video

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Genetics 002
Structure of DNA, Types of DNA grade X-IJSO.

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