Presentation- Mayo Clinic, Todd and Karen Wanek Family Program for Hypoplastic Left Heart Syndrome
Nearly 2000 infants are born each year with Hypoplastic Left Heart Syndrome and require immediate surgical intervention -- these children remain at risk for a heart transplant throughout their lives. The Todd and Karen Wanek Family Program for Hypoplastic Left Heart Syndrome (HLHS) is a purpose-driven operation forged within an academic medical environment that is designed to accelerate idealism into practice. This entity emerged out of the innovative spirit of Mayo Clinic, dedicated participation of HLHS patients and families, and the focused milestone-based vision of its philanthropic partner. The principle objective of the program is to delay or prevent heart transplant by applying functional regenerative strategies for children with HLHS. Presented by: Timothy J. Nelson, MD, PhD Director of Regenerative Medicine Consult Service, Transplant Center, Director of the Todd and Karen Wanek Family Program for Hypoplastic Left Heart Syndrome, Mayo Clinic Mindy Rice Program Manager, Todd and Karen Wanek Family Program for Hypoplastic Left Heart Syndrome, Mayo ClinicFrom:FasterCuresViews:4 0ratingsTime:28:28More inNonprofits Activism

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Presentation- Mayo Clinic, Todd and Karen Wanek Family Program for Hypoplastic Left Heart Syndrome - Video

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.Rejuvenare Stem Cell Therapy 888-988-5456
Rejuvenare Autologous Adult Stem Cell Therapy welcome video asks and answers questions on whether someone is a candidate for stem cell therapy. More can be found at http://www.rejuvenare.comFrom:rejuvenareViews:0 0ratingsTime:01:01More inPeople Blogs

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.Rejuvenare Stem Cell Therapy 888-988-5456 - Video

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"Arthritis of ankle is being treated with cell therapy at the Center for Regenerative Medicin," according to A.J. Farshchian MD, a regenerative medicine practitioner at The Center for regenerative medicine.

(PRWEB) December 02, 2012

The ankle joint is known as a diarthrodial joint. Of all the joints in the body it is probably the joint most resistant to a degenerative condition. This immunity to arthrosis is primarily associated to the joints liberal distribution of forces throughout a series of interlinked compartments. The ankle joint and accompanying foot joints support the body as would a tri-pod supporting a camera. The supporting surfaces of the ankle and foot have a tri-pod structure to its form. The ankle joint represents the lateral (or outside) support column of the tri-pod supporting form. The heel bone represents the posterior column of the support form. The mid-foot and the forefoot represent the anterior support column. This tri-pod support form is not static but dynamic in its function. As the stresses change and the strains converge on points along the weight-bearing surfaces of the ankle foot adjustments are made to maintain the center of gravity within the supporting tri-pod columns.

Rarely would age related degenerative changes be seen in the ankle and foot. When degenerative changes develop in the ankle or the other support columns it is normally preceded by a history of trauma. This trauma is usually a fracture in one of the supporting hard tissues. Serious ligamental injury can also affect a degenerative condition. Following a ligamental or fracture injuries an uncoupling of the local traumatic region occurs. This uncoupling reduces the normal cellular metabolic response to weight-bearing forces of the local traumatized area. The traumatized area is isolated from the nutritious effect of tolerable strains and exposed to harmful stresses. These harmful stresses initiate cellular destruction which later becomes a degenerative condition.

The Center for Regenerative Medicine in Miami, Florida concentrates on helping arthritic and injured people to get back to a functional level of life and their activities using non-surgical techniques and Orthopedic medicine. The center's expertise is in treatment of conditions of spine, knees, shoulders and other cartilage damages. They have developed non-surgical and rehabilitation techniques focused on treatment and management of joint pain. Their team includes health professionals organized around a central theme. Their website is http://www.arthritisusa.net

Marty Eugene http://www.arthritisusa.net 305 8668384 Email Information

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Dr. Farshchian: Arthritis of the Ankle is Being Treated with Cell Therapy at the Center for Regenerative Medicine

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Public release date: 2-Dec-2012 [ | E-mail | Share ]

Contact: Rachel Salis-Silverman Salis@email.chop.edu 267-426-6063 Children's Hospital of Philadelphia

Using powerful gene-analysis tools, researchers have discovered mutations in two related genes, ARID1A and ARID1B, that are involved in the most aggressive form of the childhood cancer neuroblastoma. While these findings do not immediately improve clinical treatments, they identify a novel pathway that is defective in these cancers, a pathway that scientists can now study to develop potential new therapies.

"These gene alterations were not previously known to be mutated in neuroblastoma, and they may significantly advance our knowledge of the underlying biological pathways that drive this disease," said study leader Michael D. Hogarty, M.D., a pediatric oncologist at The Children's Hospital of Philadelphia. "These two genes function in a group of genes that seems to play an important role in neural cell behavior, and we will now work to discover if this insight may open up new treatments for children with tumors having these mutations."

Hogarty, along with Victor Velculescu, M.D., Ph.D., of the Johns Hopkins Kimmel Cancer Center, co-led the study that appeared today in Nature Genetics.

The scientists received over $1 million in funding from the St. Baldrick's Foundation, a volunteer-driven and donor-centered charity dedicated to raising money for childhood cancer research.

The current study employed sophisticated next-generation sequencing technology that identified the entire DNA sequence for a set of neuroblastoma tumors. "When this project started, it was the first of its kind to focus on a childhood tumor," said Hogarty. "This is important, because cataloguing all the DNA mutations in neuroblastoma, or any tumor, will allow us to better understand the enemy, and ultimately to make better treatment decisions."

Striking the peripheral nervous system, neuroblastoma usually appears as a solid tumor in the chest or abdomen of young children. It accounts for 7 percent of all childhood cancers, but 10 to 15 percent of all childhood cancer-related deaths.

In the current study, Hogarty and colleagues identified alterations in two genes, ARID1A and ARID1B, neither of which had previously been reported to be involved in neuroblastoma. Both genes are thought to affect chromatin, a combination of DNA and protein that regulates the activities of genes and ultimately controls the behavior of a cell. During normal development, neural cells switch from a primitive, rapidly dividing state (neuroblasts) into a more differentiated, or mature state (neurons).

However, said Hogarty, mutations in ARID1A and ARID1B may prevent this orderly transition, keeping the neural cells in the uncontrolled stage of growth that becomes a cancerous tumor. "Unfortunately, children with these mutations have a particularly aggressive, treatment-resistant form of neuroblastoma," he added. The current study found that ARID1A and ARID1B mutations occur in 5 to 15 percent of high-risk neuroblastomas, but the pathway these genes affect may have a broader role in the diseasea possibility that Hogarty and colleagues plan to investigate further. It is possible that children having tumors with these mutations will receive more aggressive or more experimental treatments in the future.

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New gene-sequencing tools offer clues to highest-risk form of a childhood cancer

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Researchers have discovered a new gene they say helps explain how humans evolved from chimpanzees.

The gene, called miR-941, appears to have played a crucial role in human brain development and could shed light on how we learned to use tools and language, according to scientists.

A team at the University of Edinburgh compared it to 11 other species of mammals, including chimpanzees, gorillas, mice and rats.

The results, published in Nature Communications, showed that the gene is unique to humans.

The team believe it emerged between six and one million years ago, after humans evolved from apes.

Researchers said it is the first time a new gene carried by humans and not by apes has been shown to have a specific function in the human body.

Martin Taylor, who led the study at the Institute of Genetics and Molecular Medicine at the University of Edinburgh, said: 'As a species, humans are wonderfully inventive - we are socially and technologically evolving all the time.

'But this research shows that we are innovating at a genetic level too.

'This new molecule sprang from nowhere at a time when our species was undergoing dramatic changes: living longer, walking upright, learning how to use tools and how to communicate.

'We're now hopeful that we will find more new genes that help show what makes us human.'

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'Unique' evolution gene find hailed

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(MENAFN - ProactiveInvestors - Australia) Benitec Biopharma (ASX:BLT) research on muscular dystrophy shows encouraging in-vitro results via gene silencing.

The research targets the silencing of a gene (PABPN1), the mutant form of which causes the disease.

Data collected supports the development of a vector for combined silencing of the mutant gene by ddRNAi and replacement with a normal gene.

The data confirms that the delivery of a unique multi-shRNA cassette, Pabparna, to cells expressing the target gene is highly effective at silencing the target gene in those cells, reducing the expression levels to around 10%.

If achieved in vivo, this level of silencing by the clinical candidate is likely to result in significant improvement of the muscle disease, currently untreatable.

The researchers expect the optimal therapeutic to be based on both suppression of the target mutant gene and the replacement with a healthy gene.

Successful results at the stage of in vivo testing will be crucial to committing to advance the program into the clinic.

The program is being collaborated with Professor George Dickson from the Royal Holloway, University of London, and Dr Capucine Trollet from the Institut de Myologie.

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Benitec Biopharma makes breakthrough in gene silencing to treat muscular dystrophy

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Washington, December 3 (ANI): A large, international consortium of scientists has identified fifteen new genetic regions associated with coronary artery disease- taking a significant step forward in understanding the root causes of this deadly disease.

The new research brings the total number of validated genetic links with heart disease discovered through genome-wide association studies to 46.

Coronary artery disease is the process by which plaque builds up in the wall of heart vessels, eventually leading to chest pain and potentially lethal heart attacks. It is the leading cause of death worldwide.

The study, which includes researchers at the Stanford University School of Medicine, provides insights into the molecular pathways causing coronary artery disease, which is also known as coronary atherosclerosis.

"Perhaps the most interesting results of this study show that some people may be born with a predisposition to the development of coronary atherosclerosis because they have inherited mutations in some key genes related to inflammation," said Themistocles (Tim) Assimes, MD, PhD, a Stanford assistant professor of medicine and one of the study's lead authors.

"There has been much debate as to whether inflammation seen in plaque buildup in heart vessels is a cause or a consequence of the plaques themselves. Our network analysis of the top approximately 240 genetic signals in this study seems to provide evidence that genetic defects in some pathways related to inflammation are a cause," Assimes noted.

More than 170 researchers were involved in this massive meta-analysis combining genetic data from more than 190,000 research participants. Interestingly, about a quarter of the genetic regions associated with coronary disease or heart attack were also found to be strongly associated with cholesterol, especially high levels of the so-called bad cholesterol known as LDL. Another 10 percent were associated with high blood pressure. Both of these conditions are known risk factors for coronary artery disease.

"The signals that do not point to known risk factors may be pointing to novel mechanisms of disease. It is imperative that we quickly gain a better understanding of how these regions are linked to heart disease, as such understanding will greatly facilitate the development of new drugs to prevent heart disease," Assimes said.

"Studies like this one help provide new pathways for scientists to investigate in more detail. The promise is in providing better insights into the pathophysiology of this disease," added co-author Thomas Quertermous, MD, a Stanford professor of medicine.

The study is being published online Dec. 2 in Nature Genetics. (ANI)

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15 new genes associated with heart disease identified

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ScienceDaily (Dec. 2, 2012) Fifteen new genetic regions associated with coronary artery disease have been identified by a large, international consortium of scientists -- including researchers at the Stanford University School of Medicine -- taking a significant step forward in understanding the root causes of this deadly disease. The new research brings the total number of validated genetic links with heart disease discovered through genome-wide association studies to 46.

Coronary artery disease is the process by which plaque builds up in the wall of heart vessels, eventually leading to chest pain and potentially lethal heart attacks. It is the leading cause of death worldwide.

The study, published online Dec. 2 in Nature Genetics, provides insights into the molecular pathways causing coronary artery disease, which is also known as coronary atherosclerosis.

"Perhaps the most interesting results of this study show that some people may be born with a predisposition to the development of coronary atherosclerosis because they have inherited mutations in some key genes related to inflammation," said Themistocles (Tim) Assimes, MD, PhD, a Stanford assistant professor of medicine and one of the study's lead authors. "There has been much debate as to whether inflammation seen in plaque buildup in heart vessels is a cause or a consequence of the plaques themselves. Our network analysis of the top approximately 240 genetic signals in this study seems to provide evidence that genetic defects in some pathways related to inflammation are a cause."

More than 170 researchers were involved in this massive meta-analysis combining genetic data from more than 190,000 research participants. Interestingly, about a quarter of the genetic regions associated with coronary disease or heart attack were also found to be strongly associated with cholesterol, especially high levels of the so-called bad cholesterol known as LDL. Another 10 percent were associated with high blood pressure. Both of these conditions are known risk factors for coronary artery disease.

"The signals that do not point to known risk factors may be pointing to novel mechanisms of disease," Assimes said. "It is imperative that we quickly gain a better understanding of how these regions are linked to heart disease, as such understanding will greatly facilitate the development of new drugs to prevent heart disease."

Genome-wide association studies, or GWAS, were first introduced in 2005 as a way of quickly scanning the entire genome to identify differences in the DNA code, or "polymporphisms," that predispose people to various common but genetically complex diseases. Results of these studies have shown that conditions such as heart disease involve the combined, subtle effects of far more polymorphisms than initially expected, requiring multiple massive meta-analyses such as this one to reliably uncover all of these genetic signals. The hope of scientists is that by working together in ongoing worldwide collaborations, the entire genetic contribution to the cause of heart disease will eventually be identified.

"Studies like this one help provide new pathways for scientists to investigate in more detail," said co-author Thomas Quertermous, MD, a Stanford professor of medicine. "The promise is in providing better insights into the pathophysiology of this disease."

This meta-analysis study built upon previous research published last year in Nature Genetics. In that study, investigators examined 2.5 million SNPs (genetic variants at specific locations on individual chromosomes) from 14 GWA studies, which led to the discovery of 13 new gene regions associated with heart disease. Investigators looked at data from the complete genetic profiles of more than 22,000 people of European descent with heart disease and more than 64,000 healthy people.

In the new study, scientists used all information from last year's study then added to it, reaching 41,513 patients with heart disease and 65,919 control patients. To genetically fingerprint a large number of subjects in a very cost-effective manner, the researchers used a specialized genetic chip that incorporated only the top signals from the original meta-analysis of the initial 14 GWA studies.

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Inflammation may be a cause of plaque buildup in heart vessels

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Public release date: 2-Dec-2012 [ | E-mail | Share ]

Contact: Tracie White traciew@stanford.edu 650-723-7628 Stanford University Medical Center

STANFORD, Calif. Fifteen new genetic regions associated with coronary artery disease have been identified by a large, international consortium of scientists including researchers at the Stanford University School of Medicine taking a significant step forward in understanding the root causes of this deadly disease. The new research brings the total number of validated genetic links with heart disease discovered through genome-wide association studies to 46.

Coronary artery disease is the process by which plaque builds up in the wall of heart vessels, eventually leading to chest pain and potentially lethal heart attacks. It is the leading cause of death worldwide.

The study, which will be published online Dec. 2 in Nature Genetics, provides insights into the molecular pathways causing coronary artery disease, which is also known as coronary atherosclerosis.

"Perhaps the most interesting results of this study show that some people may be born with a predisposition to the development of coronary atherosclerosis because they have inherited mutations in some key genes related to inflammation," said Themistocles (Tim) Assimes, MD, PhD, a Stanford assistant professor of medicine and one of the study's lead authors. "There has been much debate as to whether inflammation seen in plaque buildup in heart vessels is a cause or a consequence of the plaques themselves. Our network analysis of the top approximately 240 genetic signals in this study seems to provide evidence that genetic defects in some pathways related to inflammation are a cause."

More than 170 researchers were involved in this massive meta-analysis combining genetic data from more than 190,000 research participants. Interestingly, about a quarter of the genetic regions associated with coronary disease or heart attack were also found to be strongly associated with cholesterol, especially high levels of the so-called bad cholesterol known as LDL. Another 10 percent were associated with high blood pressure. Both of these conditions are known risk factors for coronary artery disease.

"The signals that do not point to known risk factors may be pointing to novel mechanisms of disease," Assimes said. "It is imperative that we quickly gain a better understanding of how these regions are linked to heart disease, as such understanding will greatly facilitate the development of new drugs to prevent heart disease."

Genome-wide association studies, or GWAS, were first introduced in 2005 as a way of quickly scanning the entire genome to identify differences in the DNA code, or "polymporphisms," that predispose people to various common but genetically complex diseases. Results of these studies have shown that conditions such as heart disease involve the combined, subtle effects of far more polymorphisms than initially expected, requiring multiple massive meta-analyses such as this one to reliably uncover all of these genetic signals. The hope of scientists is that by working together in ongoing worldwide collaborations, the entire genetic contribution to the cause of heart disease will eventually be identified.

"Studies like this one help provide new pathways for scientists to investigate in more detail," said co-author Thomas Quertermous, MD, a Stanford professor of medicine. "The promise is in providing better insights into the pathophysiology of this disease."

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International study points to inflammation as a cause of plaque buildup in heart vessels

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Pamela AlliDoisWin Hospitalized on 11/28 | RAW Footage About Her Medical Condition Sarcoidosis
http://www.PamelaAlliDoisWinJessie.com 678.723.5193 Pamela AlliDoisWin Jessie Hospitalized on 11 | RAW Footage About Her Medical Condition Sarcoidosis Pamela in the hospital and being transparent and sharing a little bit about her medical conditions Sarcoidosis and Diabetes. This is RAW footage with NO MAKE UP, NO WIG, Definitely a bad hair day...Just Pamela sharing a little education on her health. Our family has 5 women in our family suffering from Sarcoidosis. Sarcoid normally affects 2 or more organs in your body. For me it has affected my lungs,eyes,legs and the glands in my mouth. Pamela AlliDoisWin Jessie Hospitalized on 11/28 | RAW Footage About Her Medical Condition Sarcoidosis What is Sarcoidosis? Sarcoidosis (pronounced SAR-COY-DOE-SIS) is an inflammatory disease that can affect almost any organ in the body. It causes heightened immunity, which means that a person #39;s immune system, which normally protects the body from infection and disease, overreacts, resulting in damage to the body #39;s own tissues. The classic feature of Sarcoidosis is the formation of granulomas, microscopic clumps of inflammatory cells that group together (and look like granules, hence the name). When too many of these clumps form in an organ they can interfere with how that organ functions. The fact that a person is more likely to develop the disease if someone in his or her close family has the disease strongly suggests that genetics plays a role. Researchers have not discovered the genes for ...From:Pamela AlliDoisWin JessieViews:48 2ratingsTime:06:01More inPeople Blogs

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Pamela AlliDoisWin Hospitalized on 11/28 | RAW Footage About Her Medical Condition Sarcoidosis - Video

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Genetics.Inheritance Part 1.mp4
How different genes and alleles are made and how they are passed on.From:Luke DeViews:1 0ratingsTime:13:41More inEducation

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Genetics.Inheritance Part 1.mp4 - Video

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The Genetics of Milk Production in Dairy Cattle
This video details the genetics of milk production in dairy cattle at a college level. It is based on various research and information learned in our Animal Science genetics class at the University of Connecticut. Enjoy! Filmed at the University of Connecticut Kellogg Dairy Center Form more information, visit http://www.animalscience.uconn.edu Special thanks to our wonderful genetics professor Dr. White, the KDC staff and of course, the cows!From:Emma PriceViews:2 1ratingsTime:05:20More inScience Technology

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The Genetics of Milk Production in Dairy Cattle - Video

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Lot 16 Hannafords Stud Natanya - Auctions Plus Progressive Genetics Sale
Lot 16 Hannafords Stud Natanya - Auctions Plus Progressive Genetics Sale Lovely natured mare broken in and quiet to ride. Has a foal at foot by Spinis Roc Star AQHA Q-37062. Not rejoined, a filly from Natanya is being retained in the stud More Information: localmedia.auctionsplus.com.au http://www.landmark-classic.com.au Sale on Auctions Plus 10th December 2012From:LandmarkEquineViews:2 0ratingsTime:00:47More inTravel Events

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Lot 16 Hannafords Stud Natanya - Auctions Plus Progressive Genetics Sale - Video

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Lot. 20 Hannafords Stud Celebration - Auctions Plus Progressive Genetics Sale
Lot No. 20 Hannafords Stud Celebration - Landmark Equine Auctions Plus Progressive Genetics Sale This beautiful mare is out of Smarty, who won 10 open drafts. Smarty also won the cut-out average at World Championship Campdraft at Sydney Show and is also the dam of Acres Little Craftsman. By sale time will have foaled to Touchwood Dippenaar ASHS 182070. Riverdale Recruit was a placegetter at Warwick and was an outstanding drafter. More Information: localmedia.auctionsplus.com.au http://www.landmark-classic.com.au Sale on Auctions Plus 10th December 2012From:LandmarkEquineViews:2 0ratingsTime:00:48More inSports

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Lot. 20 Hannafords Stud Celebration - Auctions Plus Progressive Genetics Sale - Video

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Lot 15. Hannafords Stud Skipolena- Auctions Plus Progressive Genetics Sale
Lot 15 Hannafords Stud Skipolena- Auctions Plus Progressive Genetics Sale This mare had a cut on her forearm which left a scar and an indentation in the muscle. Skipolena #39;s dam got round money at her only appearance at Warwick. This family of horses are super trainable and athletic. Skipolena is being offered with a filly foal at foot by One Moore Daddy AQHA Q-54632, giving the foal a double cross of the great One Moore Spin. More Information: localmedia.auctionsplus.com.au http://www.landmark-classic.com.au Sale on Auctions Plus 10th December 2012From:LandmarkEquineViews:0 0ratingsTime:00:47More inSports

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Lot 15. Hannafords Stud Skipolena- Auctions Plus Progressive Genetics Sale - Video

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little mix DNA
tumblr: onedirectionslittlemixer.tumblr.com twitter: twitter.com feel free to request anything! LYRICS: Perrie: Does he tell you he love you when you least expect it? Does he flutter your heart when he kisses your neck? no scientist or biology its obvious when hes holding me its only natural that im so affected Jade: Ohh and my heaert wont beat again if i cant feel him in my veins no need to question i already know All: its in his DNA DDD DNA its in his DNA and he just take my breath away BBB-Breath away i see it in his face and thats what makes a man not hard to understand perfect in every way i see it in his face nothing more to say its in his DDDD-DNA Jesy: Its the blue in his eyes that helps me see the future fingerprints that leave me covered for days yeah hey yeah now i dont have any 1st degree but i know what he does to me no need to work it out its so familiar ohhhh oh oh Jade: and my heart wont beat again if i cant feel him in my veins no need to question i already know All: iits in his DNA DDDDNA its in his DNA and he just takes my Breath away BBBBreath away i see it in his face and thats what makes a man not hard to understand perfect in every way i see it in his face nothing more to say its in his DDDDDNA Leigh Ann: Its all about his kiss contaminates my lips our energy connects its simple genetics im the x to his y its the color of his eyes he can do no wrong no he dont need to try made from the best he passes all the test got my hear beating fast its cardiac ...From:OneDirection9500Views:1 0ratingsTime:04:01More inMusic

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BIMS 320 Epigenetics Movie
BIMS 320 Epigenetic Group Project By: Texas A M Students Thank you to: http://www.youtube.com for the 18 things about Genetics portionFrom:Megan BrinkmanViews:1 0ratingsTime:12:59More inScience Technology

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Learning to read Palms
I will continue to research the topic of chiromancy. Please share your thoughts and what you know about this topic in the comments below. 4:00 I begin to read (the book). The phrase "the future is in your hands" may begin to mean more to you than you previously thought. Nowadays the practice of chiromancy is widely used by specialists in the field of genetics and psychiatry as one of the additional methods of diagnostics. " It would seem that from the very beginning there were two traditions. The first linked chiromancy closely with astrology and so produced a quasi-systematic framework for its references and predictions. The second was not connected with astrology at all, but with intuition, whose methodological principles are not clear. In the Middle Ages the Christian chiromantics found a scriptural basis for chiromancy in Job 37:7: "He sealeth up the hand of every man, that all men may know his work" which could be interpreted to mean that the hand imprints are made by God for the purpose of chiromancy." When you know yourself you are better able to . . . 9:55 predestination and free will are not imcompatible Our destiny is in our hands. 11:30 A study of palmistry can make us more optimistic Preparing to read your palm. 12:20 we chage our destiny when we change ourselves Palmistry can be used as a pleasant party past time. When you begin to study hands you begin to gain insight about the people around you. Studying the hands of a person can lead to increased intimacy ...From:marcus86edViews:1 0ratingsTime:23:34More inPeople Blogs

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Genetics basic
Science - Year 10 - Genetics Q1From:ProblemSolving PenguariumViews:0 0ratingsTime:02:01More inEducation

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Genetics basic - Video

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Lot 21. Hannafords Crown Prince- Auctions Plus Progressive Genetics Sale
Lot 21. Hannafords Crown Prince- Auctions Plus Progressive Genetics Sale This colt is the real deal. He has the looks and the breeding, his dam is by Acres Destiny, out of Tassas Robin Hood #39;s dam. Destinys Gold Dust is a full sister to Cal Gibson #39;s mare who finalled at Warwick, as well as being a full sister in blood in Serene Acres More Information: localmedia.auctionsplus.com.au http://www.landmark-classic.com.au Sale on Auctions Plus 10th December 2012From:LandmarkEquineViews:0 0ratingsTime:00:46More inSports

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Lot 18. Hannaford Forever Babe - Auctions Plus Progressive Genetics Sale
Lot 18. Hannaford Forever Babe - Auctions Plus Progressive Genetics Sale Beautiful temperament and body type, typical of her deceased sire. Has bred some beautiful foals for us. Will have a foal by sale time to Hannafords Chief Justice AQHA Q-74413, ASHS C2-199215. who is by Acres Destiny. More Information: localmedia.auctionsplus.com.au http://www.landmark-classic.com.au Sale on Auctions Plus 10th December 2012From:LandmarkEquineViews:3 0ratingsTime:00:57More inSports

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The Angus Report, Dec. 3, 2012: Performance Insights
What is an EPD? Angus Genetics Inc. #39;s Tonya Amen gives you the no-nonsense explanation.From:apicreativemediaViews:2 0ratingsTime:02:04More inPets Animals

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THE MIRACLE OF EVOLUTION
The word "evolution" is an accordion-word that can be expanded or contracted to suit the occasion. The evolutionary biologist Francisco Ayala points out that the word "evolution" can be used to mean at least three different things: 1. The process of change and diversification of living things over time. It is in this sense that biologists say that evolution is a fact. But obviously this fact, so stated, is innocuous and would not be disputed even by the most fundamentalist Young Earth Creationist. 2. Reconstruction of evolutionary history, showing how various lineages branched off from one another on the universal tree of life. 3. The mechanisms which account for evolutionary change. Darwin appealed to natural selection operating on random variations in living things in order to explain the adaptedness of organisms to their environment. With the development of modern genetics, genetic mutations came to supplement the Darwinian mechanism of natural selection by supplying an explanation for the variations on which natural selection works. Accordingly, we can call this hypothesis "neo-Darwinism." Evolution in the senses of (2) and (3) is not an established fact, despite what is said and believed in popular culture. According to Ayala, "The second and third issues mdash;seeking to ascertain evolutionary history as well as to explain how and why evolution takes place mdash;are matters of active scientific investigation. To reconstruct evolutionary history, we have to know how the ...From:POTTERS HOUSE CHRISTIAN FELLOWSHIP CHRISTCHURCHViews:2 0ratingsTime:01:56More inTravel Events

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Celebrating IR100k at IRRI!
Also see playlist of other videos from this ceremony at http://www.youtube.com On 29 November 2012, the IRRI community celebrated the100,000th rice breeding cross at IRRI, a true milestone in the Institute #39;s 52-year history existence. The parents for the 100000th cross were IR09M105 and NSICRC 214.The objective of this particular cross was to improve eating quality. Making opening comments at the event were Eero Nissila, head of Plant Breeding, Genetics and Biotechnology at IRRI -www.irri.org - and Glenn Gregorio, senior scientist/plant breeder - http://www.irri.org Alvaro Pamplona, senior research manager, demonstrates how the 100000th cross was made--as are all crosses at IRRI--for more on how the cross is made, see http://www.youtube.com Bas Bouman (right) director of the Global Rice Science Partnership (GRiSP) and Bruce Tolentino, deputy director general for communications and partnerships, "lift" the ribbon at the display showing the mature plants resulting from the cross along with the parents in the background.From:irrivideoViews:3 0ratingsTime:02:06More inEducation

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SALT LAKE CITY, Nov. 30, 2012 (GLOBE NEWSWIRE) -- Myriad Genetics, Inc. (MYGN) reported today that the Supreme Court of the United States granted certiorari agreeing to hear the case of The Association for Molecular Pathology, et al., v. Myriad Genetics, Inc., et al (Docket No. 12-398). The Supreme Court will review the earlier decision by the U.S. Court of Appeals for the Federal Circuit, which declared that Myriad's composition of matter claims covering isolated DNA of the BRCA 1 and BRCA 2 genes are patent-eligible under Section 101 of the United States Patent Act.

"Two previous decisions by the Federal Circuit Court of Appeals confirmed the patentability of our groundbreaking diagnostic test that has helped close to one million people learn about their hereditary cancer risk," said Peter Meldrum, President and CEO of Myriad Genetics. "Myriad devoted more than 17 years and $500 million to develop its BRACAnalysis(R) test. The discovery and development of pioneering diagnostics and therapeutics require a huge investment and our U.S. patent system is the engine that drives this innovation. This case has great importance for the hundreds of millions of patients whose lives are saved and enhanced by the life science industry's products."

About Genetic Patents and Genetic Testing

The BRACAnalysis test detects the presence of the BRCA1 and BRCA2 genetic mutations that can help determine a patient's risk of breast and ovarian cancer and inform treatment options. Women who test positive using Myriad's BRACAnalysis have an 82 percent higher risk of developing breast cancer and a 44 percent higher risk of ovarian cancer in their lifetimes. The test is widely available for all women with a family history of cancer and is cost effective for both patients and the healthcare system. Approximately one million women have already benefited from taking the BRACAnalysis test.

Myriad supports research studies on BRCA1, BRCA2 and other genes. More than 18,000 scientists have studied the BRCAgenes and published more than 9,000 research papers, making these genes among the most widely researched genes in history. In addition, Myriad has facilitated research through a partnership with the National Institutes of Health and provided at-cost testing for nearly 6,000 researchers receiving NIH grants.

Health economic studies conclude that Myriad's genetic tests are fairly priced. Excerpts supporting this conclusion include the following:

Additionally, Myriad has also established a Financial Assistance Program, which provides coverage at no charge to low-income patients who lack insurance. Over the past three years alone, more than 5,000 people have received free BRACAnalysis testing from Myriad. Approximately 95% of all patients in the United States have access to BRACAnalysis either through private insurance, Medicare or Medicaid. The average out-of-pocket cost to a patient is less than $100.

Even though Myriad automatically retests all positive results to confirm the findings at no additional charge to the patient, second-opinion testing is also available for all patients. Since 1999, many laboratories have performed confirmatory and second-opinion testing. The NCBI Genetic Testing Registry lists seven laboratories in the United States that conduct second-opinion testing on the BRCA 1 gene and six laboratories that test the BRCA 2 gene.

About Myriad's Patent Landscape

Myriad's intellectual property for its BRACAnalysis(R) test is strong, with 24 issued patents and more than 500 claims. This provides Myriad with extensive patent protection through 2018.

Read more from the original source:
Supreme Court of the United States to Hear Isolated DNA Patent Case

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