Public release date: 12-Nov-2012 [ | E-mail | Share ]

Contact: Zoe Tzanev ztzanev@liebertpub.com 914-740-2100 Mary Ann Liebert, Inc./Genetic Engineering News

New Rochelle, NY, November 12, 2012Mary Ann Liebert, Inc., publishers is proud to launch the Liebert Author Advocacy Program (LAAP), an innovative membership program for academic, corporate, and funding institutions designed to support Open Access (OA) and enhance the visibility and share-ability of academic research. The LAAP and associated AuthorCite portal provide a cost-effective way to support OA publishing for affiliated researchers, as well as a dedicated platform to archive, promote, and share OA articles published in any Liebert journal.

LAAP member institutions receive a 25% discount on OA article processing charges (APC) for research published in any Liebert journal; a customized microsite to highlight published OA articles; immediate global access to peer-reviewed OA articles on major indexing services; and complimentary access to the AuthorCite platform for OA authors publishing in Liebert journals. AuthorCite is a robust online marketing platform that enables authors to build professional visibility and enhance the impact of their research. Participating LAAP institutions are also eligible for a 10% discount on the subscription list price of any Liebert journal purchased directly from the publisher. LAAP participants pay a flat annual membership fee based on the number of full-time faculty and/or researchers at their institution.

"We are committed to flexible, innovative publishing services that facilitate Open Access to original research articles, alongside value-added premium journal content," says Mary Ann Liebert, president of Mary Ann Liebert, Inc., publishers. "The launch of our Liebert Author Advocacy Program serves to advance research discovery, scholarly exchange, and, ultimately, the impact of original research contributions."

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For more information on LAAP membership, visit the website at http://www.authoradvocacy.com or contact laap@liebertpub.com.

About the Publisher

Mary Ann Liebert, Inc., publishers is a privately held, fully integrated media company universally acknowledged for publishing authoritative peer-reviewed journals in the most promising areas of biomedical research, the life sciences, medicine, surgery, and public health. Mary Ann Liebert, Inc. publications continue to make critical contributions in advancing research and facilitating collaboration throughout the world in academia, industry, and government, and are also highly respected resources for legislators, policy makers, and educators. The firm publishes more than 70 journals, books, and newsmagazines. A complete list is available on our website at http://www.liebertpub.com.

Mary Ann Liebert, Inc. 140 Huguenot St., New Rochelle, NY 10801-5215 Phone: (914) 740-2100 (800) M-LIEBERT Fax: (914) 740-2101 http://www.liebertpub.com

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New Liebert Author Advocacy Program (LAAP) promotes scholarly publishing

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If you could know whether your unborn child was going to be born with a genetic defect, would you want that information? Thatisthe basis of a quad test, offered to every pregnant woman, the first in a series of detection tools to determine if something is off with the fetus.

In part one of this three part series on genetic selection, 7News looks into how this test works and the conflict between nature and science.

When Melanie and Rick Sarro of Lake Charlesdecided to start a family later in life, they knew it carried a risk for pregnancy problems, but that did notdeter their plans. "We knew that we wanted to have children and that we were gonna have to not waste any time to do it," said Melanie.

Before the Sarros celebrated their third anniversary, Zachary was here and baby number two was on the way. "The pregnancywent very smoothly, it was uneventful," said Rick, "so it was a joyous event for both of them."

Because of Melanie's age, 37, her doctor offered a standard quad test, something she did.

Dr. Marshall St. Amant is the Director of Maternal Fetal Medicine at Woman's Hospital in Baton Rouge and his team of physicians travels to Lake Charles a few times a week to treat local women with high risk pregnancies. He says asimple blood test canshow a woman's risk for genetic defects. "Itnever tells you for sure that the baby has a problem or does not have the problem you're looking for," he said, "it just says the risk is greater than or less than her predetermined risk by her age."

If the quad test does show that the mother is at risk of having a child with a genetic abnormality, then an amniocentesis is offered under the guidance of ultrasound technology with athin needle. "The fluid is removed over a period of a couple of minutes and then it's sent to a laboratory for a very specialized analysis,"said Dr. St. Amant.

Amniocentesis is the gold standard for diagnosing genetic disorders, analyzing every chromosome. "It will identify any numerical abnormality of the chromosomes, being a whole extra copy of a chromosome. It will also identify broken chromosomes," said Dr.St. Amant.

Defects are reported on a caryotype with nearly 100 percent accuracy.

The most common findings are on chromosome 18, known as Edwards syndrome and on 21, Down syndrome. "The patient would be offered the ability to continue the pregnancy," said Dr. St.Amant, "a secondary alternative would be the patient would be offered the ability to end the pregnancy."

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Genetic testing of a fetus offered for pregnant women

Recommendation and review posted by Bethany Smith

Washington, November 13 (ANI): A new study has found a genetic link between chronic pancreatitis and alcohol consumption.

Researchers from the University of Pittsburgh School of Medicine and more than 25 other health centers across the United States found a genetic variant on chromosome X near the claudin-2 gene (CLDN2) that predicts which men who are heavy drinkers are at high risk of developing chronic pancreatitis.

"The discovery that chronic pancreatitis has a genetic basis solves a major mystery about why some people develop chronic pancreatitis and others do not," said David C. Whitcomb, M.D., professor of medicine, cell biology and physiology, and human genetics at the University of Pittsburgh School of Medicine and lead author of the report.

"We also knew there was an unexpected higher risk of men developing pancreatitis with alcohol consumption, but until now we weren't sure why. Our discovery of this new genetic variant on chromosome X helps explain this mystery as well," he stated.

Chronic pancreatitis is a progressive inflammatory disease characterized by abdominal pain and permanent damage to the pancreas.

Most studies report excessive alcohol consumption as the major risk factor for adult-onset chronic pancreatitis. However, according to Dr. Whitcomb, who also is chief of the Division of Gastroenterology, Hepatology and Nutrition, only 3 percent of individuals who are alcoholics develop chronic pancreatitis, suggesting a pancreas-specific risk factor.

The study was conducted over 10 years and involved more than 2,000 patients, all of whom underwent DNA testing in a study funded by the National Institutes of Health. Researchers discovered that there was a common DNA variant on the X chromosome that is present in 26 percent of men without pancreatitis, but jumps to nearly 50 percent of men diagnosed with alcoholic pancreatitis.

Women have two X chromosomes, so most women with the high-risk DNA variant on one X chromosome appear to be protected from alcoholic chronic pancreatitis by the other X chromosome, if it is normal. Men have one X chromosome and one Y chromosome, so if they inherit a high-risk X chromosome, there is no protection.

The factor on chromosome X does not appear to cause pancreatitis, but if pancreatic injury occurs for any reason such as gallstone pancreatitis or abdominal trauma, it is more likely that the person will develop chronic pancreatitis - especially if they also drink alcohol.

"This information is important because the high-risk chromosome can be identified in patients who drink and have early signs of pancreatic injury," said Dhiraj Yadav, M.D., M.P.H., associate professor of medicine, Division of Gastroenterology, Hepatology and Nutrition at Pitt, and a co-investigator on the study.

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How alcohol consumption ups risk of chronic pancreatitis

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The Institute of Medicine of the National Academy of Sciences has elected 70 new members including Lynda Chin, Stephen Quake, and Daniel Kastner. Chin is currently a professor and chair of genomic medicine and scientific director of the Institute for Applied Cancer Science at the University of Texas MD Anderson Cancer Center. Quake is a professor of bioengineering at Stanford University and an investigator at the Howard Hughes Medical Institute. Kastner is the scientific director of the National Human Genome Research Institute, where, among other duties, he leads the inflammatory disease section of the medical genetics branch.

Caprotec Bioanalytics has appointed Jonathan Turner to be CEO and managing director. Turner will take over the CEO spot from company founder Hubert Koester, who will continue to work with the company as acting chief scientific officer and chairman of the scientific advisory board. Turner formerly was senior VP at XL Techgroup, a technology developer and equity firm, and he held senior management posts at Boehringer Ingelheim, Astrazeneca, and Schering.

Bill Bowen has been appointed by Sequenom SVP and general counsel. He will report directly to Chairman and CEO Harry Hixson and will be responsible for the company's legal and patent issues. Bowen was previously with Gen-Probe, where he was SVP and general counsel. Before that he was a business litigation partner at Luce Forward Hamilton & Scripps.

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Foundation Medicine, AstraZeneca to ID Genetic Mutations for Cancer Drug Development

Recommendation and review posted by Bethany Smith

SAN DIEGO, Nov. 12, 2012 /PRNewswire/ --Sequenom, Inc. (SQNM), a life sciences company providing innovative diagnostic testing and genetic analysis solutions, today announced its wholly-owned subsidiary, Sequenom Center for Molecular Medicine (Sequenom CMM), presented results from a study of its RetnaGene AMD laboratory-developed test to predict risk of disease progression during the 2012 Joint Meeting of the American Academy of Ophthalmology and the Asia-Pacific Academy of Ophthalmology in Chicago.

This Sequenom CMM laboratory-developed genetic test (LDT) combines patient disease stage with patient genetic variation to evaluate the risk of a patient with early or intermediate AMD to progress to advanced choroidal neovascularization (CNV) disease within 2, 5, and 10 years. CNV is the most common form of 'wet' advanced age-related macular degeneration (AMD), in which new blood vessels in the eye leak fluid, compromising central vision. Advanced disease impacts approximately 10 percent of AMD patients, but is associated with 90 percent of vision loss in AMD.

The clinical validation of the laboratory test predicting progression to CNV was conducted using patient DNA samples made available through the National Eye Institute's Age-Related Eye Disease Study (AREDS). More than 2,000 patients were genotyped for 13 single nucleotide gene polymorphisms (SNPs) in genes previously shown to be associated with CNV. Sequenom CMM compared the predictive value of a phenotype model, based on the assessment of disease grade currently used in clinical practice. The predictive model that combined genotype with phenotype was found to be more accurate in predicting CNV progression (AUC=0.96) than the phenotype model alone based on disease grade (AUC=0.89), concluding that inclusion of the genotype assessment is more effective in predicting CNV progression compared with phenotype alone.

"Physicians today rely on an assessment of patient disease stage to predict the risk of progressing to CNV, and this genetic laboratory developed test will help improve the accuracy of prediction by assessing individual risk based on the genetic predisposition of the patient," said Allan T. Bombard, M.D., Sequenom's Chief Medical Officer.

The study was conducted in compliance with the Coriell Cell Repositories Institutional Review Board, in accordance with Department of Health and Human Services (45 CFR Part 46). The dataset used for the analysis was obtained from the National Eye Institute-Age-Related Eye Disease Study (NEI-AREDS) Genetic Repository. Funding support for AREDS was provided by the National Eye Institute grant N01-EY-0-2127, National Institutes of Health, Bethesda, Maryland.

At the meeting: Paper 30031594: Combining Genotype and Phenotype to Predict Progression to Choroidal Neovascularization (CNV) in Patients with AMD (Presented Monday, November 12, 3:42 PM in S406B)

About SequenomSequenom, Inc. (SQNM) is a life sciences company committed to improving healthcare through revolutionary genetic analysis solutions. Sequenom develops innovative technology, products and diagnostic tests that target and serve discovery and clinical research, and molecular diagnostics markets. The company was founded in 1994 and is headquartered in San Diego, California. Sequenom maintains a Web site at http://www.sequenom.com to which Sequenom regularly posts copies of its press releases as well as additional information about Sequenom. Interested persons can subscribe on the Sequenom Web site to email alerts or RSS feeds that are sent automatically when Sequenom issues press releases, files its reports with the Securities and Exchange Commission or posts certain other information to the Web site.

Sequenom CMM, LLCSequenom Center for Molecular Medicine (Sequenom CMM), a CAP accredited and CLIA-certified molecular diagnostics laboratory, is developing a broad range of laboratory-developed tests with a focus on prenatal and ophthalmic diseases and conditions. These laboratory-developed tests provide beneficial patient management options for obstetricians, geneticists, maternal fetal medicine specialists, retinal specialists and ophthalmologists. Sequenom CMM is changing the landscape in genetic disorder diagnostics using proprietary cutting edge technologies.

Forward-Looking Statements Except for the historical information contained herein, the matters set forth in this press release are forward-looking statements within the meaning of the "safe harbor" provisions of the Private Securities Litigation Reform Act of 1995, including statements regarding the benefits or expectations of Sequenom CMM's genetic AMD test including the test's ability to help improve the accuracy of predicting the risk of a patient progressing to CNV by assessing individual risk based on the genetic predisposition of the patient, Sequenom's commitment to improving healthcare through revolutionary genetic analysis solutions, and Sequenom CMM changing the landscape in genetic disorder diagnostics. These forward-looking statements are subject to risks and uncertainties that may cause actual results to differ materially, including the risks and uncertainties associated with Sequenom's ability to develop and commercialize new technologies and products and to scale up its operations to meet increased product demand, particularly for new technologies and products such as Sequenom CMM's prenatal and other diagnostics testing services, Sequenom's ability to manage its existing cash resources or raise additional cash resources, customer demand, Sequenom's ability to obtain payor reimbursement and payment collection and the timing thereof, for Sequenom CMM's diagnostic test services including the MaterniT21 PLUS LDT, Sequenom's ability to convert to accrual accounting for its diagnostic test services including the MaterniT21 PLUS LDT, competition, intellectual property protection and intellectual property rights of others, government regulation particularly with respect to diagnostic products and laboratory developed tests, obtaining or maintaining regulatory approvals, ongoing litigation, including patent litigation asserting infringement by our products or challenging the validity of our patents, and other risks detailed from time to time in Sequenom, Inc.'s most recent Quarterly Report on Securities and Exchange Commission Form 10-Q and Annual Report on Securities and Exchange Commission Form 10-K and other documents subsequently filed with or furnished to the Securities and Exchange Commission. These forward-looking statements are based on current information that may change and you are cautioned not to place undue reliance on these forward-looking statements, which speak only as of the date of this press release. All forward-looking statements are qualified in their entirety by this cautionary statement, and Sequenom, Inc. undertakes no obligation to revise or update any forward-looking statement to reflect events or circumstances after the issuance of this press release.

(Logo: http://photos.prnewswire.com/prnh/20040415/SQNMLOGO)

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Study Shows Sequenom CMM's RetnaGene LDT Accurately Predicts Risk of Progression to Wet Form of AMD

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Public release date: 12-Nov-2012 [ | E-mail | Share ]

Contact: Sarah Avery sarah.avery@duke.edu 919-660-1306 Duke University Medical Center

DURHAM, N.C. In the first broad genetic landscape mapped of a Burkitt lymphoma tumor, scientists at Duke Medicine and their collaborators identified 70 mutations, including several that had not previously been associated with cancer and a new one that was unique to the disease.

Findings from the genetic sequencing of Burkitt lymphoma, an aggressive form of lymphoma, could be used to develop new drugs or aim existing therapies at mutations known to be susceptible. The researchers published their findings online Sunday, Nov. 11, 2012, in the journal Nature Genetics.

"This study lays out the most common genetic alterations in the disease, and allows us to understand the biology of the disease so we can design better therapies," said Sandeep S. Dave, M.D., MBA, MS, associate professor at Duke and senior author of the study.

Dave and colleagues sequenced the first complete Burkitt lymphoma genome, plus the genes from 59 additional Burkitt cases and 94 diffuse large B cell lymphomas, which share many of the same characteristics of Burkitt lymphoma. Similarities between the malignancies can often lead to mistaken diagnoses and failed treatments.

The researchers reported striking differences in the gene mutation patterns of Burkitt lymphomas vs. the diffuse large B cell lymphomas.

"It's important that doctors make the right diagnosis for Burkitt lymphoma, which can be cured with the correct therapies," Dave said. "But if misdiagnosed and given the standard chemotherapy regimes for diffuse large B cell lymphomas, Burkitt lymphoma patients invariably relapse."

The analysis identified 70 genes that were frequently mutated in the Burkitt lymphomas, including a number of genes that were identified in cancer for the first time. One of the newly identified gene mutations, ID3, appeared in 34 percent of the Burkitt cases, but was not evident in any of the diffuse large B cell lymphomas. The mutation has a silencing effect on a gene that suppresses cell growth, enabling cells to multiply.

Dave said this alteration alone may not cause cancer, but when it occurs along with the MYC gene mutations that are common in Burkitt lymphoma and other malignancies, it works like an accelerant to fuel tumor growth. That finding could prove helpful for developing a new drug to function like a normal ID3 gene and suppress cancer cell proliferation in lymphomas as well as numerous other cancers.

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Duke Medicine news -- Genome sequencing of Burkitt Lymphoma reveals unique mutation

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ScienceDaily (Nov. 12, 2012) A new study published online November 12 in Nature Genetics reveals a genetic link between chronic pancreatitis and alcohol consumption. Researchers from the University of Pittsburgh School of Medicine and more than 25 other health centers across the United States found a genetic variant on chromosome X near the claudin-2 gene (CLDN2) that predicts which men who are heavy drinkers are at high risk of developing chronic pancreatitis.

This finding enables doctors to identify people with early signs of pancreatitis or an attack of acute pancreatitis who are at very high risk for progressing to chronic pancreatitis, allowing them to take preventative action to slow the development of the disease, and give the pancreas a chance to heal. Once an individual develops pancreatitis it takes several years for the pancreas to deteriorate.

"The discovery that chronic pancreatitis has a genetic basis solves a major mystery about why some people develop chronic pancreatitis and others do not," said David C. Whitcomb, M.D., professor of medicine, cell biology and physiology, and human genetics at the University of Pittsburgh School of Medicine and lead author of the report. "We also knew there was an unexpected higher risk of men developing pancreatitis with alcohol consumption, but until now we weren't sure why. Our discovery of this new genetic variant on chromosome X helps explain this mystery as well."

Over 100,000 Americans suffer from chronic pancreatitis, a progressive inflammatory disease characterized by abdominal pain and permanent damage to the pancreas. Most studies report excessive alcohol consumption as the major risk factor for adult-onset chronic pancreatitis. However, according to Dr. Whitcomb, who also is chief of the Division of Gastroenterology, Hepatology and Nutrition, only 3 percent of individuals who are alcoholics develop chronic pancreatitis, suggesting a pancreas-specific risk factor.

The study was conducted over 10 years and involved more than 2,000 patients, all of whom underwent DNA testing in a study funded by the National Institutes of Health. Researchers discovered that there was a common DNA variant on the X chromosome that is present in 26 percent of men without pancreatitis, but jumps to nearly 50 percent of men diagnosed with alcoholic pancreatitis. Women have two X chromosomes, so most women with the high-risk DNA variant on one X chromosome appear to be protected from alcoholic chronic pancreatitis by the other X chromosome, if it is normal. Men have one X chromosome and one Y chromosome, so if they inherit a high-risk X chromosome, there is no protection.

The factor on chromosome X does not appear to cause pancreatitis, but if pancreatic injury occurs for any reason such as gallstone pancreatitis or abdominal trauma, it is more likely that the person will develop chronic pancreatitis -- especially if they also drink alcohol.

"This information is important because the high-risk chromosome can be identified in patients who drink and have early signs of pancreatic injury," said Dhiraj Yadav, M.D., M.P.H., associate professor of medicine, Division of Gastroenterology, Hepatology and Nutrition at Pitt, and a co-investigator on the study. "If pancreatic injury and acute pancreatitis occur, patients must stop drinking immediately."

Nationally, 16 percent of men drink alcohol at levels defined by the National Institute on Alcohol Abuse and Alcoholism as high risk. Twenty-six percent of these men who drink heavily are at high risk of chronic pancreatitis following pancreas injury. Only 10 percent of women drink alcohol at dangerous levels, and of these only 6 percent have the X chromosome variant on both X chromosomes.

"Previous discoveries show that chronic pancreatitis without alcohol involvement has a strong genetic link. This helps to eliminate the previous stigma that patients with chronic pancreatitis must also be heavy drinkers," added Dr. Whitcomb. "This study proves that there is a genetic element to the disease."

Referrals of at-risk patients are welcome at UPMC and other large academic centers. The Pancreas Clinic within the UPMC Digestive Disorder Center is designed to evaluate patients using genetic and other data to provide treatment that is individualized to each patient. In addition to clinical care, the physician-scientists who staff this clinic are actively involved in teaching physicians and trainees the art and science of personalized medicine for chronic pancreatitis.

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Genetic link between pancreatitis and alcohol consumption

Recommendation and review posted by Bethany Smith

Public release date: 12-Nov-2012 [ | E-mail | Share ]

Contact: Cristina Mestre MestreCA@upmc.edu 412-586-9776 University of Pittsburgh Schools of the Health Sciences

PITTSBURGH, Nov. 12, 2012 A new study published online today in Nature Genetics reveals a genetic link between chronic pancreatitis and alcohol consumption. Researchers from the University of Pittsburgh School of Medicine and more than 25 other health centers across the United States found a genetic variant on chromosome X near the claudin-2 gene (CLDN2) that predicts which men who are heavy drinkers are at high risk of developing chronic pancreatitis. This finding enables doctors to identify people with early signs of pancreatitis or an attack of acute pancreatitis who are at very high risk for progressing to chronic pancreatitis, allowing them to take preventative action to slow the development of the disease, and give the pancreas a chance to heal. Once an individual develops pancreatitis it takes several years for the pancreas to deteriorate.

"The discovery that chronic pancreatitis has a genetic basis solves a major mystery about why some people develop chronic pancreatitis and others do not," said David C. Whitcomb, M.D., professor of medicine, cell biology and physiology, and human genetics at the University of Pittsburgh School of Medicine and lead author of the report. "We also knew there was an unexpected higher risk of men developing pancreatitis with alcohol consumption, but until now we weren't sure why. Our discovery of this new genetic variant on chromosome X helps explain this mystery as well."

Over 100,000 Americans suffer from chronic pancreatitis, a progressive inflammatory disease characterized by abdominal pain and permanent damage to the pancreas. Most studies report excessive alcohol consumption as the major risk factor for adult-onset chronic pancreatitis. However, according to Dr. Whitcomb, who also is chief of the Division of Gastroenterology, Hepatology and Nutrition, only 3 percent of individuals who are alcoholics develop chronic pancreatitis, suggesting a pancreas-specific risk factor.

The study was conducted over 10 years and involved more than 2,000 patients, all of whom underwent DNA testing in a study funded by the National Institutes of Health. Researchers discovered that there was a common DNA variant on the X chromosome that is present in 26 percent of men without pancreatitis, but jumps to nearly 50 percent of men diagnosed with alcoholic pancreatitis. Women have two X chromosomes, so most women with the high-risk DNA variant on one X chromosome appear to be protected from alcoholic chronic pancreatitis by the other X chromosome, if it is normal. Men have one X chromosome and one Y chromosome, so if they inherit a high-risk X chromosome, there is no protection.

The factor on chromosome X does not appear to cause pancreatitis, but if pancreatic injury occurs for any reason such as gallstone pancreatitis or abdominal trauma, it is more likely that the person will develop chronic pancreatitis especially if they also drink alcohol.

"This information is important because the high-risk chromosome can be identified in patients who drink and have early signs of pancreatic injury," said Dhiraj Yadav, M.D., M.P.H., associate professor of medicine, Division of Gastroenterology, Hepatology and Nutrition at Pitt, and a co-investigator on the study. "If pancreatic injury and acute pancreatitis occur, patients must stop drinking immediately."

Nationally, 16 percent of men drink alcohol at levels defined by the National Institute on Alcohol Abuse and Alcoholism as high risk. Twenty-six percent of these men who drink heavily are at high risk of chronic pancreatitis following pancreas injury. Only 10 percent of women drink alcohol at dangerous levels, and of these only 6 percent have the X chromosome variant on both X chromosomes.

"Previous discoveries show that chronic pancreatitis without alcohol involvement has a strong genetic link. This helps to eliminate the previous stigma that patients with chronic pancreatitis must also be heavy drinkers," added Dr. Whitcomb. "This study proves that there is a genetic element to the disease."

Excerpt from:
Genetic link between pancreatitis and alcohol consumption, says Pitt team

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The Truth About Loose Skin - Is It Loose Skin or Fat?
Like / Share / Subscribe! It #39;s all greatly appreciated! Loose Skin Vs. Body Fat The cause of loose skin may change from person to person and its ability to go back to normal will be based on a variety of factors. This often happens after a drastic change in weight, commonly when weight is lost to quickly. Is it possible to non-surgically reduce loose skin? Well that is going to depend on genetics, age, amount of weight lost etc. This video will cover a few of those topics and my experience! Facebook: http://www.facebook.com Twitter: twitter.com Instagram: @mattyfusaro FusaroFitness Mailing Address: Matty Fusaro PO BOX 1746 Rocky Point, NY 11778 ***Message about monetization: This is an original video made by me and I own rights to all the content. I created this video with my own camera and used the editing software by Apple Final Cut Pro X. It contains no movies or tv visuals. I own all the pictures in it. There are no video games or performances. Outro Music *Message about Music from LDUK About LDUK Music LDUK Youtube Channel - http://www.youtube.com Song Title - Triangle DreamsFrom:Matty FusaroViews:1374 114ratingsTime:05:21More inSports

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The Truth About Loose Skin - Is It Loose Skin or Fat? - Video

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Steveo #39;s Ultimate Genetics Pack
This is a nice collection of coral for sale from Steve Connor at 828-638 zero seven seven nine.From:WestIntlvideoViews:3 0ratingsTime:00:29More inPets Animals

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Steveo's Ultimate Genetics Pack - Video

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Creatures 4 - GangNorn Style (PC, MAC, iPad, iPhone)
http://www.creatures4-thegame.co.uk What is Creatures 4? All of us have heard of artificial intelligence... Well, Creatures is artificial life. Since its release in 1996, the saga has charmed and amazed adults and children alike with its believable and lifelike characters and open-ended and in-depth gameplay. Ten years and over two million games sold later, Fishing Cactus and Bigben Interactive work together to produce a brand new game based on this amazing license. The idea is to keep what made the success of the previous games, while opening it to a wider audience. The new opus will be a Free-to-Play, available on PC / Mac, iOS (iPhone, iPad, iPod Touch) and Android systems. A collector #39;s edition full of surprises will also be available. Creatures 4 is a life simulation game featuring the most adorable creatures: the Norns. Thanks to its one of a kind Artificial Intelligence, they come with their own fully complete DNA, making them unique and allowing them to evolve, learn, interact and even reproduce! Their physical appearance and behaviors are all influenced by their genetics (but not gene manipulation!) and experience. Nature or nurture, the possibilities in Creatures 4 are absolutely infinite! Creatures 4 has a number of tools for raising Norns, such as a diagnostic system that is simple, clear, and complete. And don #39;t forget that in Creatures 4, you are not just in charge of an animal: your Norns are born, live, learn, reproduce, and die, leaving the world to the ones ...From:SmoogleViews:0 4ratingsTime:00:52More inGaming

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Creatures 4 - GangNorn Style (PC, MAC, iPad, iPhone) - Video

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Laser Genetics
Get a great deal here redirect.viglink.com?key=1f0527e04637dcdb26bf99b11836bfbf out=http%3A%2F%2Fwww%2Eamazon%2Ecom%2Fdp%2FB004SY2L06 Product Description Laser Genetics The ND-3X40 Laser Designator is a precision optical lighting instrument using advanced green laser technology. The patented Rotary Optical Collimator allows full adjustment and control of beam diameter and intensity to focus ligh where you need it most. Rotating the collimator provides enough illumination to ligh a trail at nigh or paint a target at up to 400 yards*. Two CR123 3V batteries provide over 8 hours of continuous use (4 hours of continuous at 0 Farhenheit) *Distance will vary depending on weather and terrain. Optics used on 3.5-10X50 Rifle ScopeFrom:celine hendricksViews:0 0ratingsTime:00:53More inScience Technology

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Laser Genetics - Video

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ACCESS News: Benjamin Gregg, Author, Professor of Government at UT
Tamara and Professor Benjamin Gregg from the University of Texas discuss the role of politics, science, and genetics; the difference between human rights and civil rights; and "Life, Liberty, and the Pursuit of Happiness".From:ACCESSNewsUSViews:3 0ratingsTime:27:00More inNews Politics

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ACCESS News: Benjamin Gregg, Author, Professor of Government at UT - Video

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An Introduction to Genetics
I great short video to show in your classroom to introduce Students to genetics. I thought of this video based on Georgia Perfomance Standard S7L3.From:smadrid1187Views:1 0ratingsTime:03:21More inEducation

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An Introduction to Genetics - Video

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Compact Scope
Get a great deal here redirect.viglink.com?key=1f0527e04637dcdb26bf99b11836bfbf out=http%3A%2F%2Fwww%2Eamazon%2Ecom%2Fdp%2FB005CYX8QC Product Description Compact Scope Product Description Objective 32mm Magnification: 1.5x-5x Tube Diameter: 1" Parallax Setting: 100 Reticle Type: Range Finder Field of View (ft @ 100 yds): 32.0-11.0 Eye Relief (in): 3.3 Exit Pupil (mm): 7.0-3.6 Weight (oz): 15.5 Length (in): 13.0 Battery: CR2032 3V W/E Click Value @ 100 yds 1/4"OverviewSuperior multi-coated optics sets the sniper line of scopes apart from the competition without costing you an arm and a leg. Comparable if not the same as the manufacturing process to that of the Japanese and German optics. A hint of yellowish tint can be found on most Chinese optics offered by companies such as NcStar, Aimsports, Leapers, and Barska. Sniper line of scope is crystal clear and full of useful features such as quick windage/elevation locking adjustment plate.Sniper offers not a limited lifetime warranty (as stated, you are limited to conditions in fine prints) but a 100% satisfaction guaranty on the workmanship and against any manufacturer #39;s defect.Rule the game with your new sniper scope.1.5-5x32 CQB Close Quarter Combat scope with Laser illuminating designator. Light up the night with FSI compact rifle or pistol laser illuminating designator. Instant night vision for hunting, nature watching, or SOS beacon. Rule the night with FSI laser illuminating designator. Comparable to BSA Laser Genetics.From:lucile franciscoViews:0 0ratingsTime:00:53More inScience Technology

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Compact Scope - Video

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Creatures 4 - GangNorn Style (PC, MAC, iPad, iPhone, Android - Soon!)
Instagram Sneak Peek Feed: instagram.com Norns Club @ Pinterest: pinterest.com Creatures 4 is the first and only Genetics Video Game series. After getting viewed over 1 billion times down on Earth, Psy #39;s intergalactic hit Gangnam Style has now reached Sphericus, the planet where the cute intelligent Norns race (tries to) live. Hatch eggs, breed creatures, pet them, educate them, feed them, nurture them, teach them, play with their genomes, mate them through the splicing machine or through natural means, do whatever you have to do to save the endangered species from extinction and repopulate the planet -- Tinker with evolution and balance the eco-system over generations! Each and every Norn created in the game is almost as unique as a human being: over a billion customization combination possibilities! Creatures 4 is a true Simulation of Life. Coming Spring 2013 on PC, MAC, iPad, iPhone, iPod, iPad Mini Android devices. Facebook: http://www.facebook.com Twitter: twitter.comFrom:FishingCactusGamesViews:2 0ratingsTime:00:52More inGaming

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Creatures 4 - GangNorn Style (PC, MAC, iPad, iPhone, Android - Soon!) - Video

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Seed Company Showcases New Technology
In the following video, Jacob Bates, AgriGold regional agronomist, highlights some of the company #39;s new and existing genetics that farmers can tap into for 2013.From:AgWebSaraViews:1 0ratingsTime:02:28More inEntertainment

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Seed Company Showcases New Technology - Video

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Genetic Research Project: Neurofibromatosis
Here is my presentation on Neurofibromatosis, and here are my references, shown here in the dooblydoo: Erb, MH, Uzcategui, N., See, RF, Burnstine, MA (2007). Orbitotemporal Neurofibromatosis: Classification and Treatment. Orbit, 223-228. Huijbregts, SC, de Sonneville, LM (2011). Does Cognitive Impairment Explain Behavioral and Social Problems of Children with Neurofibromatosis Type 1? Behaviour Genetics, 430-436. MedlinePlus. (2012, October 25). Neurofibromatosis. Retrieved from MedlinePlus: http://www.nlm.nih.gov Office of Communications and Public Liason. (2012, January 13). Neurofibromatosis Information Page. Retrieved from National Institute of Neurological Disorders and Stroke: http://www.ninds.nih.govFrom:boredoutofmymindizeViews:0 1ratingsTime:04:13More inEducation

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Genetic Research Project: Neurofibromatosis - Video

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Marijuana LED Grow Week 5 6 Flower - LBK KH Flowering - Germinating Poison Genetics + FREEBIES
In this update video you will be able to see week five bloom update pics, week six bloom update pics, the lady berry kush and kushy haze after a couple weeks in flower, the germination of the latest 4 strains including Poison Genetics Casey #39;s Cousin x Sour Diesel IBL. So far these growblu.com LED GROW LIGHTS have performed GREAT!!!From:ledgrowshowsViews:0 0ratingsTime:02:47More inScience Technology

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Marijuana LED Grow Week 5

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Founder effect
For more information, log on to- shomusbiology.weebly.com Download the study materials here- shomusbiology.weebly.com This video discusses the mechanism of founder effect which is an example of genetic drift. In population genetics, the founder effect is the loss of genetic variation that occurs when a new population is established by a very small number of individuals from a larger population. It was first fully outlined by Ernst Mayr in 1942,[1] using existing theoretical work by those such as Sewall Wright.[2] As a result of the loss of genetic variation, the new population may be distinctively different, both genetically and phenotypically, from the parent population from which it is derived. In extreme cases, the founder effect is thought to lead to the speciation and subsequent evolution of new species. In the figure shown, the original population has nearly equal numbers of blue and red individuals. The three smaller founder populations show that one or the other color may predominate (founder effect), due to random sampling of the original population. A population bottleneck may also cause a founder effect even though it is not strictly a new population. The founder effect is a special case of genetic drift.[3][4] In addition to founder effects, the new population is often a very small population and so shows increased sensitivity to genetic drift, an increase in inbreeding, and relatively low genetic variation. This can be observed in the limited gene pool of ...From:Suman BhattacharjeeViews:0 0ratingsTime:02:33More inEducation

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Founder effect - Video

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Gene flow
For more information, log on to- shomusbiology.weebly.com Download the study materials here- shomusbiology.weebly.com In this video lecture, I am going to talk about the gene flow among species and its importance to produce variations as well. In population genetics, gene flow (also known as gene migration) is the transfer of alleles or genes from one population to another. Migration into or out of a population may be responsible for a marked change in allele frequencies (the proportion of members carrying a particular variant of a gene). Immigration may also result in the addition of new genetic variants to the established gene pool of a particular species or population. There are a number of factors that affect the rate of gene flow between different populations. One of the most significant factors is mobility, as greater mobility of an individual tends to give it greater migratory potential. Animals tend to be more mobile than plants, although pollen and seeds may be carried great distances by animals or wind. Maintained gene flow between two populations can also lead to a combination of the two gene pools, reducing the genetic variation between the two groups. It is for this reason that gene flow strongly acts against speciation, by recombining the gene pools of the groups, and thus, repairing the developing differences in genetic variation that would have led to full speciation and creation of daughter species. For example, if a species of grass grows on both sides ...From:Suman BhattacharjeeViews:0 0ratingsTime:02:29More inEducation

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Gene flow - Video

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Flu During Pregnancy Can Lead To Autism In Child NEW STUDY
Flu During Pregnancy Can Lead To Autism In Child NEW STUDY Doctors trying to find some of the causes of autism put another piece into the puzzle on Monday: They found women who had flu while they were pregnant were twice as likely to have a child later diagnosed with autism. Those who had a fever lasting a week or longer -- perhaps caused by flu or maybe by something else -- were three times as likely to have an autistic child. The study of 96000 children in Denmark raises as many questions as it answers. But it fits in with a growing body of evidence that suggests that, in at least some cases, something is going on with a mother #39;s immune system during pregnancy that affects the developing child #39;s brain. Health officials said the finding reinforces their recommendations that pregnant women should make sure to get flu shots. Advertise | AdChoices Autism seems to be a growing problem in the United States. According to the US Centers for Disease Control and Prevention, autism spectrum disorder affects one in 88 children, including about one in 54 boys. The autism spectrum refers to a broad range of symptoms, from the relatively mild social awkwardness of Asperger #39;s syndrome to profound mental retardation, debilitating repetitive behaviors and an inability to communicate. Scientists agree that it #39;s not just a matter of better diagnosis; the numbers seem to be growing because more children are indeed developing autism. But no one is sure why. Genetics are a large factor -- if ...From:HotBlockNewsViews:0 0ratingsTime:01:30More inNews Politics

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Flu During Pregnancy Can Lead To Autism In Child NEW STUDY - Video

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Hardy Weinberg equilibrium
For more information, log on to- shomusbiology.weebly.com Download the study materials here- shomusbiology.weebly.com This video tutorial explains the Hardy-Weinberg equilibrium and its importance in population genetics studies.From:Suman BhattacharjeeViews:0 0ratingsTime:17:18More inEducation

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Hardy Weinberg equilibrium - Video

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Star Visitors - Dr. Richard Boylan - Coast to Coast AM Classic
http://www.jetnews.us Date: 02-03-11 Host: George Noory Guests: Dr. Richard J. Boylan, Joshua P. Warren Appearing in the second hour, anthropologist specializing in Star Cultures, and certified clinical hypnotherapist, Dr. Richard Boylan shared updates on ET visitors and #39;star children. #39; According to his information, a powerful ruling cabal plans to conduct a fake alien invasion, to divert attention away from their oppressive tactics. The recent UFO sighting in Jerusalem is an example of this, he added. Humans were bio-engineered by ETs 275000 years ago, and they continue to upgrade human genetics, with some 96% of grade school children being "star seeds" or "star children," he cited. There are some 1483 separate alien races who have visited Earth, including two groups that reside here-- the Tall Whites in Nevada, and the Saami people who came from Barnard #39;s star, and now live above the Arctic Circle, he claimed. Opens Lines Joshua P. Warren The latter half of the show featured Open Lines, with callers sharing accounts that included encounters with small aliens, and the Old Hag. Paranormal investigator Joshua P. Warren joined George to field the calls in the last hour, and related how he was saved during an angelic intervention as a young boy. He also commented that humans may be developing a new sensory organ that will allow them to view astral entities around us. Biography: Dr. Richard J. Boylan is a Ph.D. behavioral scientist, anthropologist, University associate professor ...From:C2CPlanetViews:24 1ratingsTime:01:55:01More inEducation

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Star Visitors - Dr. Richard Boylan - Coast to Coast AM Classic - Video

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Why descendants of "Black African Eve" look so different? (Evolution biology)
Human skin color is primarily due to the presence of melanin in the skin. Skin color ranges from almost black to white with a pinkish tinge due to blood vessels underneath. Variation in natural skin color is mainly due to genetics, although the evolutionary causes are not completely certain. According to scientific studies, natural human skin color diversity within populations is highest in Sub-Saharan African populations,[2] with skin reflectance values ranging from 19 to 46 (med. 31) compared with European and East Asian populations which have skin reflectance values of 62 to 69 and 50 to 59 respectively. The term "range" is loosely defined in this case, as African albinos have obviously not been taken into consideration when calculating the "range". The natural skin color can be darkened as a result of tanning due to exposure to sunlight. The leading theory is that skin color adapts to intense sunlight irradiation to provide partial protection against the ultraviolet fraction which produces damage and thus mutations in the DNA of the skin cells. Other factors that can modify skin color include protection from ambient temperature, infections, skin cancer or frostbite, an alteration in food, and sexual selection. The social significance of differences in skin color has varied across cultures and over time, as demonstrated with regard to social status and racism. About 70000--100000 years ago some modern humans began to migrate away from the tropics to the north where ...From:saddoboxing1Views:0 0ratingsTime:05:30More inScience Technology

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Why descendants of "Black African Eve" look so different? (Evolution biology) - Video

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