Orthopedic Stem Cell Therapy - Queens, New York
Benjamin Bieber MD of #CrossBayPMR in Howard Beach, New York has had great success with #stemcell therapy using your own fat cells. Avoid invasive #jointreplacement surgery and get back to...

By: Cross Bay Physical Medicine and Rehabilitation, P.C.

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Orthopedic Stem Cell Therapy - Queens, New York - Video

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Jeremy #39;s Stem cell therapy journey
This is a video about stem cells and stem cell therapy. Created by #39;Vivienne Armstrong #39;.-- Created using PowToon -- Free sign up at http://www.powtoon.com/join -- Create animated videos and...

By: Vivi Armstrong

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Jeremy's Stem cell therapy journey - Video

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York University researchers have learned how living beings can keep gene expression in check -- which might partly explain the uncontrolled gene expression found in many cancers.

"Using yeast as a model organism, we studied the Tup1 protein, a negative regulator of gene expression," says Biology Professor Emanuel Rosonina, adding, "This protein binds to some genes and blocks their expression, helping to ensure genes that shouldn't be turned on remain inactive."

The current study, jointly conducted by York University and Columbia University researchers, suggests that Small Ubiquitin-like Modifier (SUMO) modifies proteins bound to active genes, in order to prevent unfettered gene over-expression that can be harmful to the organism. "One of the ways SUMO does this is by promoting the binding of Tup1 to active genes, which then acts to reduce their expression to appropriate levels," explains Rosonina, in the Faculty of Science at York U.

"Sumoylation controls the timing of Tup1-mediated transcriptional deactivation" published March 13, 2015 in Nature Communications is a follow up to a previous study which found SUMO in every gene examined.

"As a result of the previous study, we reported that SUMO is probably important for controlling expression of active genes because we found it on every gene we looked at, but only when they were turned on," notes Rosonina.

Considering that many tumours have abnormal levels of SUMO, it will be important to examine whether inappropriate SUMO modifications in these tumours are related to the uncontrolled gene expression that is observed in most cancers, the research concludes.

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The above story is based on materials provided by York University. Note: Materials may be edited for content and length.

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Organisms can keep gene expression in check, biologist says

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1 hour ago by Geoff Vivian A Scarlet robin (Petroica boodang). Credit: Roger Smith

The scarlet robin (Petroica boodang) may be classified into separate eastern and western Australian species following thousands of years of evolution separated by natural geographical barriers including the Nullarbor Plain.

WA Museum geneticist Gaynor Dolman, who studied a dozen accepted species, says the robin is one of several southern Australian birds whose populations have been separated by natural barriers for so long they have become distinct species.

These include the chestnut quail-thrush (Cinclosoma castanotum), white-naped honeyeater (Melithreptus lunatus), New Holland honeyeater (Phylidonyris novaehollandiae) and white-eared honeyeater (Nesoptilotis leucotis).

Dr Dolman says several natural geographical barriers appear to have prevented populations from mixing at different times.

She says the most permanent and significant of these is the "Eyrean Barrier" consisting of the Flinders Ranges and the salty Lake Eyre Basin in South Australia, which has kept some populations separate for at least 600,000 years.

She attributes the oldest species divergence, perhaps 1.5 million years ago, to this barrier.

Dr Dolman says the Nullarbor Plain had been in the landscape for a long time and potentially affects the birds.

"But they also might be able to get past the Nullarbor at different periods where there's more drainage in the system or the shoreline is further back," she says

Dr Dolman the Nullarbor may have led to a later species divergence about 275,000 years ago in some cases, and there may have been a more recent divergence that is yet to be confirmed.

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Single gene points to separate bird species

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summer internship training in Genetic Engineering
CytoGene Research Development ,Lucknow provides several modules of training as well as dissertation/project in Genetic Engineering http://cytogene.in/r-DNA...

By: CytoGene Research Development

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summer internship training in Genetic Engineering - Video

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Steps in Genetic Engineering - AIIMS AIPMT State CET Botany Video Lecture

By: Rao IIT Academy

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Steps in Genetic Engineering - AIIMS AIPMT State CET Botany Video Lecture - Video

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Replacing faulty genes in early human embryos and germ cells is within our grasp. Such changes affect DNA in the nucleus and so would be heritable; ultimately, they could be used to make a genetically modified baby.

There are already reports that groups in China, the US and the biotech industry have done this kind of genetic engineering in the lab, prompting some scientists to call for a moratorium on this work. But the underlying technology is potentially hugely disruptive, offering easier and more precise ways to manipulate genes. Here's what you need to know about this new frontier in genetics.

What's behind the current controversy over human genetic engineering? Several teams have tried modifying the genome of a human embryo and submitted their results for publication, according to an article published in Technology Review on 5 March. This kind of research is already illegal in some countries.

What exactly has been done? We don't know the details yet. But based on what's been done in monkeys, the work probably involved fertilising donated human eggs by injecting a sperm and then, while the fertilised eggs were still at the one-cell stage, injecting various RNAs. These RNAs cut DNA at specific sites, tricking our natural DNA repair system into destroying or replacing one or more genes a technique known as gene editing. The embryos would then have been allowed to develop for a few days until they reached the blastocyst stage containing a few hundred cells before being destroyed. The embryos' DNA would have been sequenced to see whether the gene editing had been successful.

So it's now possible to genetically engineer humans? It was already possible. The hard part is doing it safely and efficiently. Gene editing was first developed decades ago, but it was very difficult and expensive, and often didn't work very well. To create just one genetically modified animal this way typically required hundreds of attempts. But editing genes has become a lot easier, cheaper and more efficient thanks to the CRISPR system, developed just a couple of years ago. This was the method used to modify the human embryos.

Why are some scientists calling for a moratorium? They argue that human germline gene editing is dangerous, unnecessary and would lead to designer babies. They also claim it could lead to a backlash that would impede the use of gene editing for making non-heritable changes to our bodies. A wide range of diseases, from HIV to cancer, could potentially be treated this way.

So is germline gene editing dangerous? We don't know yet. One of the points of trying it with human embryos is to find out. The main worry is so-called off-target mutations, that is, unintended changes to the genome. The monkey studies suggest the risk is low. The risk also has to be viewed in context: the DNA in our cells naturally mutates. Each of us is born with around 50 new mutations, the vast majority of which have no known effect.

Is germline gene editing the only way to prevent some heritable diseases? Severe disorders caused by a single faulty gene, such as cystic fibrosis, can usually be prevented by screening. Parents can opt for IVF, for instance, with preimplantation genetic diagnosis that is, testing the embryos before they are implanted to ensure they don't have the faulty gene. But when children are at risk of inheriting several faulty genes, screening usually isn't feasible.

Serious genetic disorders that strike early in life are, fortunately, rare. But we probably all inherit dozens if not hundreds of gene variants that increase our risk of common diseases such as cancer, dementia, depression and so on.

Will gene editing lead to designer babies? Possibly. But it's not clear that many, or any, parents, would want to pay large sums and undergo a risky process IVF has its dangers just to ensure their child is blue-eyed. And it is of course possible to allow germline editing for disease prevention while banning it for other purposes. It would be hard for anyone who broke the ban to get away with it, given that genetic tests could reveal whether a child's DNA has been altered. Some argue, though, that parents should be free to choose their child's characteristics if they want.

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Editing human embryos is genetics' new battleground

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Mower Genetics - Flush

By: Mower Genetics

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Mower Genetics - Flush - Video

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Newswise BETHESDA, MD Nearly 1,000 scientists from 35 countries will attend the 28th Fungal Genetics Conference organized by the Genetics Society of America (GSA), March 1722, 2015, at the Asilomar Conference Grounds in Pacific Grove, CA. The conference will feature approximately 900 presentations (including over 200 talks) of cutting-edge fungal genetics research with a focus on filamentous fungi on topics including genomics, gene regulation, cell biology and development, evolutionary biology, fungal-host interactions and biotechnology.

Professor Michael J. Hynes from the University of Melbourne will present the Perkins/Metzenberg Lecture on Saturday, March 21 at 5:45 pm, just prior to the closing conference banquet. This lecture is given at each Fungal Genetics Conference by a leader in fungal genetics research; it often includes an historical and thought-provoking perspective as well as an outlook on the future of the field. For more information on the conference program, including the timing of plenary sessions each of which ends with a Chair's Choice Talk presenting high impact studies nominated by the scientific community please see the schedule of events.

Numerous species of fungi are instrumental model organisms for the study of biology particularly of genetics and related phenomena including heredity, gene function, reproduction, metabolism, and evolution. For example, the field of molecular genetics is said to have begun in Neurospora crassa, where experiments showing that genes act by regulating definite chemical events led to the 1958 Nobel Prize in Physiology or Medicine. The genetic and experimental tractability of fungi make them excellent systems for studying the effects and mechanisms of mutations and transposable DNA elements that move to different locations in the genome. Their short yet complex life cycles allow for studies of long-term evolution and sexual reproduction, while their multicellularity allows for studies of development, intracellular communication, and morphogenesis. Interactions between fungi and other organisms have also enabled studies of pathogenesis, infection, and symbiotic relationships. The industrial applications of fungi are numerous, including biotechnology, biofuels, fermentation, pharmaceuticals, citric acid production, and more. The 28th Fungal Genetics Conference will integrate the areas in which fungi have been instrumental as model organisms or industrial tools.

For additional information, please see the conference website athttp://www.genetics-gsa.org/fungal/2015/.

MediaEligibility:

The 28th Fungal Genetics Conference is open to media representatives, including those frombona fideprint, broadcast, radio, and online venues, and freelance writers on a verifiable assignment from an established news source. Please contactpress@genetics-gsa.orgfor information about complimentary press registration.

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Fungal Genetics Meeting to Showcase Breakthroughs in Molecular Biology

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YOUNG gun shearers took to the board to shear 750 wethers in Australia's largest commercial evaluation of Merino genetics - the Peter Westblade Memorial Merino Challenge (PWMCC).

The PWMCC which runs from 2014 to 2016 kicked off with 50 teams of 30 wethers last year.

Once all wethers were together they were separated into two sections, with one half being lotfed with their meat measured, while the remaining 15 from each team were used for wool evaluations.

The average age of the wethers when they entered the challenge was seven-months-old, they were all shorn upon entry and last week were shorn again with 11 months wool growth.

First shearing was conducted over a two-day period at the TAFE Riverina's Primary Industries Centre at Wagga last week.

Challenge convener, Craig Wilson, Wagga, said the challenge evaluated both commercial carcase and wool attributes of randomly sampled wethers lambs from across Australia.

Preliminary results show Jerilderie's Ross and Irene Wells' Willandra Merino stud, NSW, genetics as a standout with two of their clients claiming the two top places on the board.

Wedderburn growers, Ian and Julie Gould, "Wattle Grange", Victoria, finished with the best figures for their team of wethers that had a wool average of 19.4 micron, cut 8.6kg of wool, weighed 63.6kg, had a meat value of $86.07, wool value of $67.95 and total sheep value of $154.02.

Second placed in the statistics were Maurice and Nancye Hicks, "Springfield", Cootamundra, with his 20.6 micron team of wethers, that cut 9.4kg of wool, weighed 64.2kg had a meat value of $86.92, a wool value of $66.16 and a total sheep value of $153.08.

Mr Wells said when his clients put teams in the challenge it provided an opportunity to benchmark his genetics against others breeders, and in particular the concept that Australian Sheep Breeding Values (ASBV's) and Estimated Breeding Value's (EBV's) were the only way the sheep industry could go forward.

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Merino genetics challenge kicks off

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Gene Therapy Gastrointestinal Insight: Trends and Challenges Analysed in Research Report
Gene Therapy Gastrointestinal Insight: Pipeline Assessment, Technology Trend, and Competitive Landscape provides the information across the gene therapy value chain covering gene therapy ...

By: James Jacob

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Gene Therapy Gastrointestinal Insight: Trends and Challenges Analysed in Research Report - Video

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Melbourne Conversations - Personalised Medicine: Me and My Genome
Global genomics expert Professor Michael Snyder from Stanford University sequenced his genome and got a shock! Hear his story, get the latest on the work aro...

By: Melb Conversations

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Melbourne Conversations - Personalised Medicine: Me and My Genome - Video

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Putting personalized medicine to the test
At the UAB Hugh Kaul Personalized Medicine Institute, researchers are translating cutting-edge genetic discoveries into the real world of patient care.

By: University of Alabama at Birmingham

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Fauza D: Amniotic fluid and placental stem cells.

Best Pract Res Clin Obstet Gynaecol 2004, 18:877-891. PubMedAbstract | PublisherFullText

Parolini O, Alviano F, Bagnara GP, Bilic G, Bhring HJ, Evangelista M, Hennerbichler S, Liu B, Magatti M, Mao N, Miki T, Marongiu F, Nakajima H, Nikaido T, Portmann-Lanz CB, Sankar V, Soncini M, Stadler G, Surbek D, Takahashi TA, Redl H, Sakuragawa N, Wolbank S, Zeisberger S, Zisch A, Strom SC: Concise review: isolation and characterization of cells from human term placenta: outcome of the first international workshop on placenta derived stem cells.

Stem Cells 2008, 26:300-311. PubMedAbstract | PublisherFullText

Pozzobon M, Ghionzoli M, De Coppi P: ES, iPS, MSC, and AFS cells. Stem cells exploitation for Pediatric Surgery: current research and perspective.

Pediatr Surg Int 2010, 26:3-10. PubMedAbstract | PublisherFullText

Miki T, Marongiu F, Dorko K, Ellis EC, Strom SC: Isolation of amniotic epithelial stem cells.

Curr Protoc Stem Cell Biol 2010, Chapter 1:Unit 1E 3. PubMedAbstract | PublisherFullText

Miki T, Strom SC: Amnion-derived pluripotent/multipotent stem cells.

Stem Cell Rev 2006, 2:133-142. PubMedAbstract | PublisherFullText

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Stem Cell Research & Therapy | Full text | Amnion-derived ...

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Importance Most patients with relapsing-remitting (RR) multiple sclerosis (MS) who receive approved disease-modifying therapies experience breakthrough disease and accumulate neurologic disability. High-dose immunosuppressive therapy (HDIT) with autologous hematopoietic cell transplant (HCT) may, in contrast, induce sustained remissions in early MS.

Objective To evaluate the safety, efficacy, and durability of MS disease stabilization through 3 years after HDIT/HCT.

Design, Setting, and Participants Hematopoietic Cell Transplantation for Relapsing-Remitting Multiple Sclerosis (HALT-MS) is an ongoing, multicenter, single-arm, phase 2 clinical trial of HDIT/HCT for patients with RRMS who experienced relapses with loss of neurologic function while receiving disease-modifying therapies during the 18 months before enrolling. Participants are evaluated through 5 years after HCT. This report is a prespecified, 3-year interim analysis of the trial. Thirty-six patients with RRMS from referral centers were screened; 25 were enrolled.

Interventions Autologous peripheral blood stem cell grafts were CD34+ selected; the participants then received high-dose treatment with carmustine, etoposide, cytarabine, and melphalan as well as rabbit antithymocyte globulin before autologous HCT.

Main Outcomes and Measures The primary end point of HALT-MS is event-free survival defined as survival without death or disease activity from any one of the following outcomes: (1) confirmed loss of neurologic function, (2) clinical relapse, or (3) new lesions observed on magnetic resonance imaging. Toxic effects are reported using National Cancer Institute Common Terminology Criteria for Adverse Events.

Results Grafts were collected from 25 patients, and 24 of these individuals received HDIT/HCT. The median follow-up period was 186 weeks (interquartile range, 176-250) weeks). Overall event-free survival was 78.4% (90% CI, 60.1%-89.0%) at 3 years. Progression-free survival and clinical relapse-free survival were 90.9% (90% CI, 73.7%-97.1%) and 86.3% (90% CI, 68.1%-94.5%), respectively, at 3 years. Adverse events were consistent with expected toxic effects associated with HDIT/HCT, and no acute treatment-related neurologic adverse events were observed. Improvements were noted in neurologic disability, quality-of-life, and functional scores.

Conclusions and Relevance At 3 years, HDIT/HCT without maintenance therapy was effective for inducing sustained remission of active RRMS and was associated with improvements in neurologic function. Treatment was associated with few serious early complications or unexpected adverse events.

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High-Dose Immunosuppressive Therapy and Autologous ...

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Window pow
"Goodie MoB-Cell Therapy", sound recording administered by: SME.

By: hees crazzie

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Window pow - Video

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The New Botox: Stem Cell Therapy Cream Reviews
http://buildingabrandonline.com/buildabrandwithjamalspikes/what-is-jeunesse/ The New Botox: Stem Cell Therapy Cream Reviews Stem cell therapy is the use of stem cells to treat or prevent a...

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The New Botox: Stem Cell Therapy Cream Reviews - Video

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Genetic Engineering - AIIMS AIPMT State CET Botany Video Lecture

By: Rao IIT Academy

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Genetic Engineering - AIIMS AIPMT State CET Botany Video Lecture - Video

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Genetic Engineering and Mutations - how they are connected
Genetic Engineering and Mutation video/ project. Did my best for the first time doing this so hope you enjoy!

By: yaggzy98

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Genetic Engineering and Mutations - how they are connected - Video

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Abstract: Asthma and allergic diseases are complex conditions caused by a combination of genetic and environmental factors. More than 100 genes have been associated with asthma and related conditions through candidate gene approaches, but issues of insufficient replication have made conclusions difficult to draw. Despite this, several overarching themes in the biology and pathogenesis of asthma have been revealed as a result of this work. In mid-2007, the first genome wide association study (GWAS) targeting asthma was published, and in the intervening years more than a dozen such studies have been reported examining asthma, allergic diseases, and related intermediate phenotypes and quantitative traits. A few previously suspected genetic variants have been confirmed in these studies as asthma susceptibility loci, or as loci contributing to disease severity or response to treatment. Additionally, unexpected and largely uncharacterized genes have been identified as new susceptibility loci for asthma, altering lung function or perturbing immune function. In this review, we summarize these GWAS, as well as the functional themes and characteristics underlying asthma that have been revealed through decades of genetic and genomic research.

Introduction

Asthma is a chronic inflammatory condition of the lungs, characterized by excessive responsiveness of the lungs to stimuli, in the forms of infections, allergens, and environmental irritants. During an asthma attack, lung airways will produce excess mucus and swell, and muscles around the airways will tighten leading to airway obstruction, tightness in the chest, coughing, and wheezing. Data from the National Institutes of Health suggests that 50% of U.S. asthma cases are attributable to specific allergies. Currently, 22.9 million Americans suffer from asthma, and the prevalence has increased dramatically since 1980. Asthma is the leading chronic illness in U.S. children, with 6.8 million affected in 2006 (American Lung Association, 2008). Twin studies have shown that there is a genetic element to asthma susceptibility, with heritability of the condition estimated at between 0.36 and 0.77 (Duffy et al., 1990; Harris et al., 1997; Koppelman et al., 1999; Nieminen et al., 1991). The first study to link a genetic locus (chromosome 11q13) to asthma was published in 1989 (Deichmann et al., 1999). Since then more than 100 candidate genes described in more than 1,000 publications have been found in connection to asthma or an associated phenotype, like elevated IgE levels, bronchial hyperresponsiveness, or eosinophilia.

An Overview of the Analysis of the Genetic Contributions in Asthma

Researchers have been successful in identifying the genetic underpinning of many single-gene disorders. It has been comparatively difficult to identify the genetic basis of complex genetic disorders, such as asthma, allergies, and autoimmune disease, with multifactorial inheritance and significant environmental contributors. Three study designs are routinely employed to investigate genetic contributions in complex diseases: genome-wide linkage studies, candidate gene association studies, and genome-wide association studies.

Linkage studies: Genome-wide linkage study design focuses on families affected by the disease of interest. With less genetic recombination occurring between closely related individuals, it is possible to screen the entire genome with a panel of relatively few, evenly spaced markers, searching for variants that are either unique to or over-represented in affected individuals. If such a region is found, it is said to be linked with the disease trait, and the genes within this region can become candidates for further analysis, including association study followed by positional cloning of the gene. Unlike the candidate gene association study (see below), this study design allows for the identification of genes and pathways previously not suspected of contributing to the disease in question. However, because large families of affected individuals are needed, these studies are expensive and difficult to conduct. Moreover, while they are effective at identifying genes with low frequency variants with high penetrance and large effects, they often lack the statistical power to identify genes of modest effect that are attributed to common alleles. This is in contrast to genome-wide association studies (discussed below), which are best suited to the identification of common variants with lower penetrance and smaller effects. In this way, linkage studies and association studies are used to address different questions, and are, in fact, complementary.

Approximately 20 genome-wide linkage screens have been reported in different populations investigating chromosomal regions that are linked to asthma and atopy, or related phenotypes like elevated IgE levels, wheezing, and bronchial hyperresponsiveness. A number of chromosomal regions have been repeatedly identified across multiple studies that contain genes of biological relevance to asthma and allergic disease, including the cytokine cluster on chromosome 5q [containing interleukin 3 (IL3), IL5, and granulocyte/macrophage colony-stimulating factor (GMCSF)], FCER1B on 11q, IFNG (interferon ) and STAT6 on 12q, and IL4R (the IL-4R chain, also part of the IL-13R) on 16p. Linkage studies followed by positional cloning approaches have resulted in the identification of a handful of novel asthma susceptibility genes, including CYFIP2 (Noguchi et al., 2005), DPP10 (Allen et al., 2003), HLAG (Nicolae et al., 2005), PHF11 (Zhang et al., 2003), GPRA (Laitinen et al., 2004), and ADAM33 (Van Eerdewegh et al., 2002). GPRA (G protein-coupled receptor for asthma) and ADAM33 (a disintegrin and metalloproteinase domain-containing protein 33) have generated considerable interest, as their expression in bronchial smooth muscle cells suggests roles in the pathobiology of asthma and pulmonary allergic disease (Laitinen et al., 2004).

Candidate gene studies: In a candidate gene association study, a particular gene (or set of genes) is selected for study based on its biological plausibility or suspected role in the phenotype of interest. The incidence of variants in this gene is compared between a group of individuals affected with the phenotype (cases) and a group of controls. The strength of such a design lies in the statistical power and relative ease of recruiting large cohorts, compared to family-based studies. The main limitations of such a design are its inability to identify novel or unsuspected genes and pathways contributing to the pathogenesis of a disorder, and its susceptibility to unknown population structures in cases or controls. Candidate gene association studies are best suited to identifying common genetic variants of modest effect (Risch and Merikangas, 1996).

More than 1,000 papers have been published with candidate gene studies examining asthma and related phenotypes, identifying more than 100 candidate genes. However, surprisingly few of these candidate gene discoveries have been rigorously replicated, and many have been examined and failed replication in subsequent studies. The loci identified in candidate gene studies of asthma and associated phenotypes have been extensively reviewed elsewhere (Ober and Hoffjan, 2006; Vercelli, 2008; Zhang et al., 2008). Among genes identified in candidate studies are receptors for detection of microbial products (TLRs, CD14, CARD15, among others); various cytokines and cytokine signaling proteins involved in T cell survival, proliferation, and differentiation; genes involved in lung function, development, and response to stimuli (ADRB2, CFTR, SPINK5, etc.); genes involved in epithelial barrier function and innate immunity (FLG and DEFB1) (Levy et al., 2005; Palmer et al., 2007); genes believed to be involved in the responses to environmental exposures (GSTM1, GSTP1, and GSTT1) (Halapi and Hakonarson, 2004; Hoffjan et al., 2003; Kabesch, 2005; Vercelli, 2008). Genes that have been extensively replicated include the beta2 adrenergic receptor gene (Liggett, 1995; Martinez et al., 1997; Potter et al., 1993); the cytokines, receptors, signaling proteins, and transcription factors involved in TH1 (T helper 1) and TH2 differentiation of T cells, like IL4, IL4RA, IFNG, IFNGR1, STAT6, GATA3, and TBX21 (Basehore et al., 2004; Haller et al., 2009; Munthe-Kaas et al., 2008; Pyklinen et al., 2005; Randolph et al., 2004; Suttner et al., 2009; Tantisira et al., 2004; Zhou et al., 2009); and genes involved in the cellular responses that characterize atopic disease, such as IL13 and its receptor and the FCER1B gene (Howard et al., 2002; Kabesch et al., 2006; Potaczek et al., 2009; Vladich et al., 2005; Wu et al., 2010).

Genome wide association studies: Rapid advances in microarray technology that now permit the high-throughput genotyping of hundreds of thousands of single nucleotide polymorphisms (SNPs) has allowed for the development of a third type of study, the genome-wide association study (GWAS). In this design many SNPs are compared across the entire genome between cases and controls. Like the candidate gene association study, this design facilitates the collection of a large number of cases and controls for analysis, increasing statistical power. In contrast, however, it permits a hypothesis-free search for gene variants associated with disease, revealing new targets for researchers. As mentioned above, GWAS are well-suited for discovery of common alleles with relatively small effects.

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The Genetics of Asthma and Allergic Disorders - Michael E ...

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The Sims 3 - Perfect Genetics Challenge Ep.68 Marisol the Teacher
Come join me on my latest journey into the complex world of sims 3 genetics, as I try to get perfect foals and perfect children. Will I succeed in getting pe...

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The Sims 3 - Perfect Genetics Challenge Ep.68 Marisol the Teacher - Video

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Genetics and Gregor Mendel
This clip covers Gregor Mendel #39;s work leading to our understanding of heredity. Topics covered include; Punnett Squares, probability, segregation, independent assortment, dominant and recessive...

By: Inspire The Mind

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Genetics and Gregor Mendel - Video

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Do You Have Good or Bad Muscle-Building Genetics?
Download my FREE 12 Week Program! http://musclemonsters.com/massinaflash Have fitness questions? Ask here: http://facebook.com/musclemonsters Instagram: @mus...

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Do You Have Good or Bad Muscle-Building Genetics? - Video

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The Sims 3 - Perfect Genetics Challenge - Pt8 - The Wedding
If you like this video please leave a thumbs up, it really helps Open fully for *NEW SCHEDULE* info and social media links Weekly Schedule (Subject t...

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The Sims 3 - Perfect Genetics Challenge - Pt8 - The Wedding - Video

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Genetics and Evolution HD Tutorial - Part 0: Quick Tour
Part 0 of tutorial series on Genetics and Evolution HD iPad app. Focuses on a quick tour of the entire app. Later parts in the series will focus on individua...

By: Russell M

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Genetics and Evolution HD Tutorial - Part 0: Quick Tour - Video

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