BrainStorm Cell Therapeutics

By: Alliance for Regenerative Medicine

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BrainStorm Cell Therapeutics - Video

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Cesca Therapeutics

By: Alliance for Regenerative Medicine

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Cesca Therapeutics - Video

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NeoStem

By: Alliance for Regenerative Medicine

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NeoStem - Video

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Cytori Therapeutics

By: Alliance for Regenerative Medicine

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Cytori Therapeutics - Video

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What Is Cardiac Cell Therapy?

In its simplest form, cardiac cell therapy is simply the use of stem cells to regenerate new heart tissue. Stem cells were originally used to grow your heart before you were born. Stem cells capable of growing new heart tissue reside in all of us. Through the use of trial-tested technologies, your own stem cells can be used to grow and repair your cardiac tissue.

The most difficult aspect of this therapy was developing a way to isolate your stem cells and put them to use to grow new heart tissue. And thanks to years of research, this process has been developed and tested in clinical trials with favorable results.

What Is The Procedure?

There is a wide variety of methods of placing stem cells in the body or near the organ they are intended to help. One procedure tested under trial is through the use of catheters (a specialized tube) and is being implemented in a new state-of-the-art clinic by a U.S. licensed veteran cardiologist. This process requires only a local anesthetic and minimal recovery time (hours vs days). And your own cardiologist is consulted closely to make sure you are a good candidate for the procedure and to monitor your improvements when you return home.

If you'd like our recommendations on qualified cardiac stem cell clinics, please don't hesitate to contact us at info@heartcell.org or call us at (310) 362-0562. We'd be happy to connect you to a clinic, doctor or stem cell patient for you to explore your options further.

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What Is Stem Cell Therapy | Stem Cell For Heart | Cardiac ...

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I found the whole process fascinating and rewarding, and when Alison contacted me to tell me that the first couple Id donated to hadnt eventually conceived, she also told me she was setting up Altrui, and I got involved. Its an amazing thing to be a part of. I wouldnt donate again, as Im focusing on my own family now, but I love supporting other donors with their journeys.

I told Lyndon about it all not long after we met, but there was never a problem he has two children from a previous relationship so we both come with a past. Having my daughter has just confirmed how precious my eggs must have been to the couples whose lives I have changed. Im sure that when she is able to understand what Ive done she will be proud of her mum.

Alan Fisher 35, is a data analyst and lives in Nottingham with his girlfriend, Cat. He joined the UKs blood cancer charity and bone marrow register, Anthony Nolan (anthonynolan.org), in 2010 and donated bone marrow at the London Clinic in January

It was a memorable drive to work the day I decided to donate. I tuned into the local radio station to hear a six-year-old boy hosting the breakfast show: he had leukaemia and was raising awareness for the Anthony Nolan register. It was amazing to hear a young, confident voice doing such a brave thing, and I pulled into the office car park feeling uplifted. But as I reached down to turn off the engine the show ended, and I heard the usual presenter explaining that it had been a tribute to the boy, who had died because a donor hadnt been found in time. There and then I knew I would sign up.

I went along to a Join for Joel event organised in memory of the boy, Joel Picker Spence. It was easy: all I had to do was give a saliva sample. Knowing I could be called to donate within months, years or never, I didnt think about it much after that.

A year and a half later I was contacted and told there was a potential recipient for my bone marrow, but after more tests it transpired that they didnt need me. It was a bit of an anticlimax, to be honest. But in 2013, just before Christmas, I got another phone call and recognised the number on my phone. Its my turn now, I thought.

My employers were great about me taking time off. The hospital wanted to take bone marrow under general anaesthetic from my pelvic bone. It seems like the more invasive option you can sometimes give by a stem cell blood donation but as I dont like needles I didnt mind the idea of being knocked out.

The procedure itself went fine: I spent the night before at hospital and was taken to theatre early. When I awoke after the operation, which took less than an hour, I actually thought it hadnt happened. I was left feeling drained, but only for a few days. I also had two small puncture wounds in the small of my back, but they healed nicely. For me, it was a minor inconvenience for the recipient and their family, I hope it has meant a lot more. I found out afterwards that the amount of bone marrow needed indicated that the recipient was a child. Before I was discharged, I also found out it was a young boy, about the same age as Joel.

Jay Kelly 36, is a fertility and birth hypnotherapist. She is divorced and lives in Harrogate with her four daughters, aged 13, 10 and seven (twins). She recently gave birth to a baby for another couple, whom she met through Surrogacy UK (surrogacyuk.org)

Deciding to become a surrogate wasnt some road to Damascus moment. It was something that had been bubbling under for a long time. Through my work I meet a lot of women unable to conceive and I just cant imagine how distressing it must be for them. My children are everything to me, and it struck me that if I could help a couple who couldnt have what I have, it would be a pretty amazing thing to do.

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Would you donate a kidney to someone you had never met?

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Jeunesse Luminesce Ultimate Lifting Masque
How would you like to do something once a week that will change your life and make you look and feel better about yourself? Well, this is what you do. You si...

By: allen hookens

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Jeunesse Luminesce Ultimate Lifting Masque - Video

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Irvine, CA (PRWEB) April 03, 2014

DrSkinSpa.com is a top-tier skin care web-retail store. It places its primary focus on bringing clinically tested skin care creations that are manufactured using naturally derived ingredients. The company proudly markets an extensive line of natural and effective anti wrinkle cream skin products. Skin care rejuvenators are just one of the many categories of beauty products sold here and DrSkinSpa.com has just added Eminence Bamboo Firming Fluid, 1.2 oz. to its extensive line.

The organic skin care product that is Eminence Bamboo Firming Fluid, 1.2 oz., contains an abundance of plant ingredients, essential oils, and anti-aging Swiss Green Apple Stem Cells. When placed together in this anti aging products, wrinkles and lines are smoothed, hydrated, and the skin is firmed up for a younger appearance.

The key ingredients in Eminence Bamboo Firming Fluid, 1.2 oz. include bamboo, both coconut oil and water, a natural retinol alternative complex with chicory root and tara tree, Swiss Green Apple Stem Cells, and monoi, a fragrant and firming Tahitian oil.

Bamboo has both soluble and insoluble fiber, free-radical fighting antioxidants, proteins, skin-enriching vitamins and minerals to help firm and anti age skin. Coconut oil is included in Eminence Bamboo Firming Fluid, 1.2 oz., for its moisturizing effects, which also help restore skins natural moisture barrier. This oil also works as an antioxidant. The coconut water in this serum balances the skins pH, returning moisture to skin; it also tones the complexion. Coconut water has natural reserves of Vitamin C, electrolytes, calcium, potassium and phosphorous, all plusses for both skin and body.

The Natural Retinol Alternative Complex in Eminence Bamboo Firming Fluid, 1.2 oz., is a combination of chicory root natural sugars (oligosaccharides) and tara tree. The sugars from chicory root firm up loose and sagging skin with immediate activity. It also increases collagen synthesis. Tara tree provides long-lasting moisture.

Dr. Farid Mostamand, owner of DrSkinSpa.com, says, Eminence Bamboo Firming Fluid, 1.2 oz., contains the patented PhytoCellTec. These are the Swiss Green Apple Stem Cells concentrate formula that has been clinically shown to reduce and prevent signs of aging.

DrSkinSpa.com is doctor operated and owned. The company studies and choosesfor sale only the finest products, with clinically proven and natural ingredients. DrSkinSpa.com extends to customers a two-week money-back guarantee for every product sold on their web site. The site also provides customers with a 120% price protection warranty in addition to no cost shipping. Complimentaryaesthetician consultations are also available. DrSkinSpa.com is owned by Crescent Health Center and is based in Anaheim, California.

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DrSkinSpa.com Announces the Addition of Eminence Bamboo Firming Fluid, 1.2 oz.

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A genetic tweak can make light work of some nervous disorders. Using flashes of light to stimulate modified neurons can restore movement to paralysed muscles. A study demonstrating this, carried out in mice, lays the path for using such "optogenetic" approaches to treat nerve disorders ranging from spinal cord injury to epilepsy and motor neuron disease.

Optogenetics has been hailed as one of the most significant recent developments in neuroscience. It involves genetically modifying neurons so they produce a light-sensitive protein, which makes them "fire", sending an electrical signal, when exposed to light.

So far optogenetics has mainly been used to explore how the brain works, but some groups are exploring using it as therapy. One stumbling block has been fears about irreversibly genetically manipulating the brain.

In the latest study, a team led by Linda Greensmith of University College London altered mouse stem cells in the lab before transplanting them into nerves in the leg this means they would be easier to remove if something went wrong.

"It's a very exciting approach that has a lot of potential," says Ziv Williams of Harvard Medical School in Boston.

Greensmith's team inserted an algal gene that codes for a light-responsive protein into mouse embryonic stem cells. They then added signalling molecules to make the stem cells develop into motor neurons, the cells that carry signals to and from the spinal cord to the rest of the body. They implanted these into the sciatic nerve which runs from the spinal cord to the lower limbs of mice whose original nerves had been cut.

After waiting five weeks for the implanted neurons to integrate with the muscle, Greensmith's team anaesthetised the mice, cut open their skin and shone pulses of blue light on the nerve. The leg muscles contracted in response. "We were surprised at how well this worked," says Greensmith.

Most current approaches being investigated to help people who are paralysed involve electrically stimulating their nerves or muscles. But this can be painful because they may still have working pain neurons. Plus, the electricity makes the muscles contract too forcefully, making them tire quickly.

Using the optogenetic approach, however, allows the muscle fibres to be stimulated more gently, because the light level can be increased with each pulse. "It gives a very smooth contraction," says Greensmith.

To make the technique practical for use in people, the researchers are developing a light-emitting diode in the form of a cuff that would go around the nerve, which could be connected to a miniature battery pack under the skin.

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Muscle paralysis eased by light-sensitive stem cells

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Introduction to Stem Cell Therapy
Dr. Michael Belich of Integrative Medical Clinics talks about Stem Cell Therapy basics. For more detailed information go to http://www.integrativemc.com.au.

By: Integrative Medical Clinics

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Introduction to Stem Cell Therapy - Video

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Regulated information 3 April, 2014

TiGenix licenses exclusive marketing and distribution rights for ChondroCelect to Sobi

Sobi to assume responsibility for the commercialisation of ChondroCelect in existing and new markets in Europe and beyond

Sobi's considerable expertise and resources will enhance the availability of ChondroCelect to many more patients in many more countries

TiGenix to focus its resources on developing its pipeline of allogeneic treatments using expanded adipose-derived stem cells (eASC's)

Leuven (BELGIUM) - 3 April, 2014 -TiGenix NV (Euronext Brussels: TIG), the European leader in cell therapy, announced today that it has licensed the marketing and distribution of ChondroCelect, the cell-based medicinal product for the repair of cartilage defects of the knee, to the international specialty healthcare company dedicated to rare diseases, Swedish Orphan Biovitrum AB ('Sobi', NASDAQ OMX Stockholm: SOBI).

ChondroCelect was the first cell-based product to be approved in Europe. It is currently available for patients and reimbursed in Belgium, the Netherlands and Spain. Sales of ChondroCelect in 2013 were Euro 4.3 million, a growth of 25% on a like-for-like basis over 2012.

Sobi will continue to market and distribute the product where it is currently available and has also acquired the exclusive rights to expand the product's availability to patients in multiple additional territories, including the rest of the European Union, Norway, Switzerland, Turkey, and Russia, plus the countries of the Middle East and North Africa.

TiGenix will receive a royalty of 22% of the net sales of ChondroCelect in the first year of the agreement, and 20% of the net sales of ChondroCelect thereafter. There will be no upfront or milestone payments. The agreement will take effect on 1 June 2014, and has a duration of 10 years.

"We are delighted to reach this agreement with Sobi", said Eduardo Bravo, CEO of TiGenix. "With its experience of marketing and distributing specialty products, and with its human and financial resources, Sobi has the ability to bring ChondroCelect to a far greater number of patients in many more countries. This then allows TiGenix to focus its human and financial resources on the development of its platform and pipeline of allogeneic treatments using expanded adipose-derived stem cells (eASC's) for the benefit of patients suffering from a range of inflammatory and immunological conditions."

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TiGenix : licenses exclusive marketing and distribution rights for ChondroCelect to Sobi

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New York, New York (PRWEB) April 03, 2014

Dr. Jeffrey Adler, New York podiatrist and Owner/Medical Director of Adler Footcare New York, will be interviewed by radio personality Laura Smith and taking live calls on New York talk show 77 WABC about the use of stem cells to treat chronic foot pain.

Adler Footcare New York uses live birth stem cells to help treat patients with chronic foot problems such as: Plantar fasciitis, Osteoarthritis, Achilles tendonitis and torn soft tissue. The stem cell treatment is proving to be much more effective than traditional treatments like physical therapy or orthotic therapy.

Stem cells have the ability to replicate themselves or change into the cell type that is needed to repair damaged tissue. The therapy works by directly introducing live stem cells into the affected area causing pain. Its approved by the FDA and consistently reviewed by medical professionals to remove the potential of any communicable diseases.

Dr. Adler often travels to other states to share his knowledge with other doctors in his profession. His latest research on stem cell therapy has gained much interest among his peers in the medical industry.

When compared to traditional treatments, stem therapy is proving to be more successful and longer lasting, Dr. Adler said. We are seeing patients heal much quicker and return to their normal activities much sooner.

To learn more about stem cell replacement therapy or to schedule a consultation with a New York podiatrist at Adler Footcare, call (212) 704-4310 or visit http://www.mynycpodiatrist.com.

About Adler Footcare New York Dr. Jeffrey L. Adler, Medical/Surgical Director and owner of Adler Footcare New York has been practicing podiatric medicine since 1979 and has performed thousands of foot and ankle surgeries. Dr. Adler is board certified in Podiatric Surgery and Primary Podiatric Medicine by the American Board of Multiple Specialties in Podiatry. Dr. Adler is also a Professor of Minimally Invasive Foot Surgery for the Academy of Ambulatory Foot and Ankle Surgeons. As one of only several in the country who perform minimally invasive podiatric surgery, Dr. Adlers patients enjoy significantly reduced recovery times.

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New York Podiatrist Dr. Jeffrey Adler to Be Interviewed April 5 on NewsTalkRadio 77 WABC About the Use of Stem Cell ...

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PUBLIC RELEASE DATE:

3-Apr-2014

Contact: Carrie Strehlau carrie.strehlau@stjude.org 901-595-2295 St. Jude Children's Research Hospital

(MEMPHIS, Tenn. April 3, 2014) The St. Jude Children's Research HospitalWashington University Pediatric Cancer Genome Project found mutations in the tumor suppressor gene TP53 in 90 percent of osteosarcomas, suggesting the alteration plays a key role early in development of the bone cancer. The research was published today online ahead of print in the journal Cell Reports.

The discovery that TP53 is altered in nearly every osteosarcoma also helps to explain a long-standing paradox in osteosarcoma treatment, which is why at standard doses radiation therapy is largely ineffective against the tumor. The findings follow the first whole genome sequencing of osteosarcoma, which is diagnosed in about 400 children and adolescents annually, making it the most common pediatric bone tumor.

"Osteosarcoma treatment has remained largely unchanged for more than 20 years, and cure rates are stalled at about 70 percent. This study lays a foundation for new therapies and more immediately identifies numerous mutations in TP53 missed by previous studies that did not include whole genome sequencing," said co-corresponding author Michael Dyer, Ph.D., a Howard Hughes Medical Institute investigator and member of the St. Jude Department of Developmental Neurobiology. Jinghui Zhang, Ph.D., member of the St. Jude Department of Computational Biology, is the other corresponding author.

TP53 carries instructions for assembling the p53 protein, which plays a role in DNA repair and cell death. Inactivation of p53 helps tumor cells survive radiation therapy. Previous studies estimated that TP53 was mutated in a quarter to half of osteosarcomas, suggesting that a significant proportion of patients with this tumor should respond to radiation. That was not the case, Dyer said.

"With whole-genome sequencing, we are gaining new insights into the way various mutations in TP53 promote the development of osteosarcomas," said co-author Richard K. Wilson, Ph.D., director of The Genome Institute at Washington University School of Medicine in St. Louis. "This information will be very helpful in designing treatment protocols."

The study involved whole genome sequencing of 34 osteosarcoma tumors from 32 patients. The patients' normal genomes were also sequenced.

The research revealed that 55 percent of TP53 mutations were caused by structural variations. These alterations occur when chromosomes break and are reassembled. Osteosarcoma is just the second cancer with TP53 mutations resulting from chromosomal rearrangements rather than point mutations, which are small changes in the DNA that makes up the gene. "This suggests that the cell that gives rise to osteosarcoma may either be particularly susceptible to chromosomal breaks or better able to tolerate breaks when they occur," Dyer said.

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Tumor suppressor gene TP53 mutated in 90 percent of most common childhood bone tumor

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PUBLIC RELEASE DATE:

3-Apr-2014

Contact: Kathryn Ruehle kruehle@liebertpub.com 914-740-2100 Mary Ann Liebert, Inc./Genetic Engineering News

New Rochelle, NY, April 3, 2014In many recent incidents of premeditated mass shooting the perpetrators have been male and dressed in black, and may share other characteristics that could be used to identify potential shooters before they commit acts of mass violence. Risk factors related to the antihero, dark-knight persona adopted by these individuals are explored in an article in Violence and Gender, a new peer-reviewed journal from Mary Ann Liebert, Inc., publishers. The article is available free on the Violence and Gender website at http://www.liebertpub.com/vio.

In the article "Costuming, Misogyny, and Objectification as Risk Factors in Targeted Violence," Brian Van Brunt, EdD and W. Scott Lewis, The NCHERM Group, LLC (Malvern, PA), suggest reasons why persons who commit mass shootings are drawn to dark popular culture imagery, how these cultural factors may contribute to the violence, and what risk factors could be useful to law enforcement and behavioral investigation teams seeking to identify individuals who might be preparing for an attack.

"'Objectification' of victims and 'costuming' are specific offender behaviors that will give threat assessment teams throughout the world greater insights into the motivation of mass shooters and just how ceremonial their preparations are," says Mary Ellen O'Toole, PhD, Editor-in-Chief of Violence and Gender and Senior FBI Profiler/Criminal Investigative Analyst (ret.). "The value of this information in being able to identify these offenders beforehand based on their behavior so that we can prevent future acts of mass murder is very significant."

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About the Journal

Violence and Gender is the only peer-reviewed journal focusing on the understanding, prediction, and prevention of acts of violence. Through research papers, roundtable discussions, case studies, and other original content, the Journal critically examines biological, genetic, behavioral, psychological, racial, ethnic, and cultural factors as they relate to the gender of perpetrators of violence. Led by Editor-in-Chief Mary Ellen O'Toole, PhD, Forensic Behavioral Consultant and Senior FBI Profiler/Criminal Investigative Analyst (ret.), Violence and Gender explores the difficult issues that are vital to threat assessment and prevention of the epidemic of violence. Violence and Gender is published quarterly online with Open Access options and in print, and is the official journal of The Avielle Foundation.

About the Publisher

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Dress and behavior of mass shooters as factors to predict and prevent future attacks

Recommendation and review posted by Bethany Smith

PUBLIC RELEASE DATE:

3-Apr-2014

Contact: Vicki Cohn vcohn@liebertpub.com 914-740-2100 Mary Ann Liebert, Inc./Genetic Engineering News

New Rochelle, NY, April 3, 2014Nearly 7% of Americans married between 2005-2012 met on social networking sites. How those couples compare to couples who met through other types of online meetings or the "old-fashioned" way in terms of age, race, frequency of Internet use, and other factors is explored in an article in Cyberpsychology, Behavior, and Social Networking, a peer-reviewed journal from Mary Ann Liebert, Inc., publishers. The article is available free on the Cyberpsychology, Behavior, and Social Networking website.

In "First Comes Social Networking, Then Comes Marriage? Characteristics of Americans Married 2005-2012 Who Met Through Social Networking Sites," Jeffrey Hall, PhD, University of Kansas, Lawrence, describes the characteristics that are more common among recently married individuals who met online via social networking sites (SNS).

"Facebook use grew dramatically during the 2005-2012 time period studied," says Brenda K. Wiederhold, PhD, MBA, BCB, BCN, Editor-in-Chief of Cyberpsychology, Behavior, and Social Networking, from the Interactive Media Institute, San Diego, CA. "It will be useful to continue to observe how these trends change as various groups of individuals become more frequent users of SNS," says Dr. Wiederhold.

###

About the Journal

Cyberpsychology, Behavior, and Social Networking is a peer-reviewed journal published monthly online with Open Access options and in print that explores the psychological and social issues surrounding the Internet and interactive technologies, plus cybertherapy and rehabilitation. Complete tables of content and a sample issue may be viewed on the Cyberpsychology, Behavior, and Social Networking website.

About the Publisher

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Which couples who meet on social networking sites are most likely to marry?

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Genome-wide association studies (GWAS) represent an unbiased and hypothesis-free method based on the data produced by the human HapMap Project and the fact that genetic variance at one locus can predict, with high probability, genetic variance at adjacent loci.49 As such, the human genome can be surveyed for common variants (those present in more than 5% of the population) by genotyping about 500000 accurately chosen markers, so-called tag single nucleotide polymorphisms.50 GWAS have led to the identification of thousands of loci that affect susceptibility to complex disorders, broadening understanding of the pathogenesis of many diseases.51 However, inherent drawbacks of these studies are an inability to identify sequence variants (the studies identify loci) and rare alleles, detection of non-coding variants of unknown effect, reproducibility, and, in most cases, identification of susceptibility variants that confer only a small risk of disease. In a genome-wide scan (followed by hierarchical fine mapping in a larger dataset) of six multiplex families with familial idiopathic pulmonary fibrosis from southeastern Finland, Hodgson and colleagues52 identified a shared haplotype on chromosome 4q31 that was more common in affected individuals than in healthy controls. This haplotype harboured ELMOD2, a gene expressed in alveolar macrophages and alveolar epithelium. Although ELMOD2 is a plausible candidate gene in the pathogenesis of idiopathic pulmonary fibrosis since dysfunctional ELMOD2 might potentially cause an abnormal immune response at the alveolar epithelium level,53 gene sequencing did not show any DNA variations. The first GWAS done for sporadic idiopathic pulmonary fibrosis, which examined more than 200000 SNPs in 159 Japanese patients and 934 controls,54 confirmed a previously reported association with TERT,41 substantiating the putative contribution of telomere replication and stabilisation in the pathogenesis of idiopathic pulmonary fibrosis (table). Noth and colleagues67 did a GWAS followed by two independent case-control studies in a large cohort of EuropeanAmerican patients with idiopathic pulmonary fibrosis. They identified novel genetic variants within TOLLIP (11p15.59) and SPPL2C (17q21.31) that were associated with susceptibility to idiopathic pulmonary fibrosis. Additionally, one variant within TOLLIP (rs5743890), which resulted in reduced gene expression of TOLLIP in the lungs of patients with idiopathic pulmonary fibrosis, protected against the development of fibrosis but was linked with an increased rate of disease progression and risk of mortality. TOLLIP variants seemed to confer susceptibility to idiopathic pulmonary fibrosis independently of MUC5B rs35705950, a promoter variant that significantly increases the risk of development of idiopathic pulmonary fibrosis and that resides on the same genetic locus as TOLLIP, although the two genes are separated by a recombinant hotspot.61 Whether these two genes act independently of one another has not been resolved. The role of TOLLIP in regulation of the innate immune system makes it a biologically plausible candidate in the pathogenesis of idiopathic pulmonary fibrosis. Fingerlin and colleagues68 did the largest GWAS in idiopathic pulmonary fibrosis so far. Findings of this study, which included 2492 patients with idiopathic interstitial pneumonia (most of whom had idiopathic pulmonary fibrosis) and more than 6000 controls, not only confirmed three previously known genetic associations (TERC at 3q26, TERT at 5p15, and MUC5B at 11p15) but also identified seven novel risk loci within genes involved in host defence, cellcell adhesion, and DNA repair. The association between risk of idiopathic pulmonary fibrosis and OBFC1, another gene that affects telomere length, highlights the importance of early cell senescence in the development of pulmonary fibrosis. Another gene linked to risk of pulmonary fibrosis is DSP (6p24.3), which encode desmosomes. Desmosomes are a component of adhesive intercellular junctions and are essential to preserve the integrity of tissue that undergo mechanical stress, such as the lung periphery during respiration.69 This GWAS also identified several loci with as yet unknown attributions to specific genes.

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Genetic determinants of pulmonary fibrosis: evolving concepts : The Lancet Respiratory Medicine

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Inheritance

Dr. Sharon Moalem (Grand Central Publishing)

In Town: The author will appear at the Ottawa Writers Festival on April 24 at 6:30 p.m., Knox Presbyterian Church, 120 Lisgar St.

When Californian Craig Venter sequenced his own genome in the year 2000, he wittingly started a great transformation that is now taking hold in the delivery of health care. But that change is unfolding in a global society that is not ready.

Thats the warning delivered by Dr. Sharon Moalem, a medical doctor, geneticist, businessman, inventor and author of the book Inheritance: How Our Genes Change Our Lives and Our Lives Change Our Genes.

Moalem will appear at the Ottawa Writers Festival and what he has to say is kind of mind-blowing.

First of all, because we now have the ability to sequence everyones genome and see all the markers that a person has, we can see clearly the consequences of personal choices.

For example, one person may not be able to metabolize caffeine and probably shouldnt drink a lot of coffee, while another has no problem with sugar and can drink all the soft drinks she wants.

For Moalem, who has focused on rare diseases over his career, sequencing the genome, meant that we are all rare, every one of us. Not just the 10 per cent of Americans and Canadians who have a rare condition. We all have genetic idiosyncrasies that affect who we are and how we live.

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Genetic science is transforming health care and introducing ethical dilemmas

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Savage Genetics - Dead Silence Remix (SICK BASE DROP)
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Savage Genetics - Dead Silence Remix (SICK BASE DROP) - Video

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Rastafari Shabbat #27 Tazria: Black Motherhood Law, Blood Leprosy Genetics
Rastafari Tazria Shabbat #27: Black Motherhood Law, Blood Leprosy Genetics RT LIKE, POST SHARE! WATCH [VIDEO] SUBSCRIBE, JOIN OUR SOCIAL MEDIA NETWORKI...

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Rastafari Shabbat #27 Tazria: Black Motherhood Law, Blood & Leprosy Genetics - Video

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Attack of The B-Team E16 #39; #39;Advanced Genetics #39; #39;
Join the Lapis Nation today! I hope you guys enjoyed! 5 likes? Mods =============== Advanced Genetics by ObsiLP Archimedes Ships by Balkondeur Alpha Artifice...

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Attack of The B-Team E16 ''Advanced Genetics'' - Video

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Genetics (Attack of the B-Team #11)
We return to Attack of the B-team! I know a lot of you have been waiting on this 😀 Watch more of this series! http://bit.ly/1eFfwZX Technic Launcher: http:/...

By: Cub Games

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Genetics (Attack of the B-Team #11) - Video

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Mukund Thattai - Molecular genetics
PROGRAM: School and Discussion Meeting on Population Genetics and Evolution PROGRAM LINK: http://www.icts.res.in/program/PGE2014 DATES: Saturday 15 Feb, 2014 - Monday 24 Feb, 2014 VENUE:...

By: ICTS Talks

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Mukund Thattai - Molecular genetics - Video

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UCL Genetics of Human Disease MSc

By: StudyUCL

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UCL Genetics of Human Disease MSc - Video

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Genetics of Human Disease MSc
To find out more please visit: http://www.ucl.ac.uk/ugi/education/mscgens.

By: StudyUCL

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Genetics of Human Disease MSc - Video

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NEW YORK (TheStreet) --Response Genetics (RGDX) surged 25% to $1.50 at 10:26 a.m. on Thursday after the molecular diagnostic test developer announced contracts with six new provider networks, which increases its total membership to more than 174 million U.S. members.

The contracts span 10 states and includeBlue Cross Blue Shield contracts in Arizona, Delaware, Iowa, Pennsylvania, South Dakota and West Virginia; an Independent Physician Association that covers two states in the North West region; and a Commercial Health Plan that covers two states in the North East region.

Thanks to the deals, Response Genetics is now in-network with thirteen Blue Cross Blue Shield health plans, which increases the number of Blue Cross Blue Shield subscribers with direct access to Response Genetics to approximately 23 million.

Must Read: Warren Buffett's 10 Favorite Growth Stocks

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Why Response Genetics (RGDX) Stock Is Soaring Today

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